An unprotected traveler's blood would boil at normal body temperatures because the pressure is so low. Temperatures in the mesophere decreases with altitude, because there are few gas molecules in the mesophere to absorb the sun's radiation, the heat source is the stratosphere below. The air in the mesophere has extremely low density and as a result the air pressure is very low.
Black the creamer makes it look white along with the side of the cup that it is in and the lighting and your phones picture could also make it different than it looks
<span>Antibiotic resistance arises due to the evolution of the organisms they are designed to destroy. Subsequent generations of such organisms develop resistance to the antibiotic. Hospitals' overuse of antibiotics quickens the process, as does the administration of antibiotics to feedlot cattle, whose meat is consumed by people.</span>
Answer:
B. The mutation results in a new, dominant allele
C. The mutation occurs in a gene that controls development and alters differentiation of a cell type during development.
D. The mutation occurs in a codon and alters the function of the final protein
Explanation:
All the above things will change the <u>ultimate expression</u> or phenotype by altering the proteins. Choices B, C, and D will all change the outer functioning.
Choice A only affects the rate of transcription, so it may go faster or slower, but the end product will be the same.
This part that doesn't look like it's one of the choices ("The mutation occurs in a portion of an intron not responsible for exon splicing.") would not affect phenotype, because introns are removed before the RNA is sent out.
Choice E says that the amino acid sequence is unchanged, meaning the protein final product will be the same and the expression will not change.
<span>Rhabdomyolysis constitutes a common cause of acute renal failure and presents paramount interest. A large variety of causes with different pathogenetic mechanisms can involve skeletal muscles resulting in rhabdomyolysis with or without acute renal failure. Crush syndrome, one of the most common causes of rhabdomyolysis presents increased clinical interest, particularly in areas often involved by earthquakes, such as Greece and Turkey. Drug abusers are another sensitive group of young patients prone to rhabdomyolysis, which attracts the clinical interest of a variety of medical specialties.
We herein review the evidence extracted from updated literature concerning the data related to pathogenetic mechanisms and pathophysiology as well as the management of this interesting syndrome.
Keywords: Rhabdomyolysis, acute renal failure, myoglobin, crush syndrome
The first case of the crush syndrome, which constitutes one of the main causes of rhabdomyolysis, was reported in Sicily in 1908, after an earthquake1,2. In 1930, in the Baltic area, an epidemic of myoglobinuria was observed due to consumption of contaminated fish. Interest in rhabdomyolysis and crash syndrome was stimulated during the World War II particularly after the bombing in London, where the victims developed acute renal failure and myoglobinuria1.
Rhabdomyolysis is a rupture (lysis) of skeletal muscles due to drugs, toxins, inherited disorders, infections, trauma and compression3. Lysis of muscle cells releases toxic intracellular components in the systemic circulation which leads to electrolyte disturbances, hypovolemia, metabolic acidocis, coagulation defects and acute renal failure due to myoglobin4.
The skeletal muscle consists of cylindrical myofibrils, which contain variant structural and contraction proteins. Actin and myosin, arranged in thin and thick filaments respectively, form the repeated functional units of contraction, the sarcomeres5. The sarcoplasmic reticulum constitutes an important cellular calcium storage. It is structurally connected to the t-tubules, that are formed by invaginations of the muscle cell plasma membrane, the sarcelemma, around every fibril (Figure 1). After the sarcelemma depolarization, the stimulation arrives, through the t-tubules junctions, at the sarcoplasmic reticulum, inducing the calcium ions release and triggering muscle contraction6.</span>
