It's important because it is easy to apply it too tightly and impede circulation. Allows for smoother fit and prevents gaping caused by variation in contour of the limb.
Answer:
Hyperimmunoglobulinemia E Syndrome
Explanation:
Hyperimmunoglobulinemia E Syndrome or Job syndrome is an immunodeficiency disorder in which the white blood cells are incapable of producing an inflammatory response to infectious organisms. It is an autosomal dominant disorder. It is characterized by recurring bacterial infections, specially of lung and skin. There are also other symptoms like abromalities in vascular or skeletal system and elevated IgE level.
Symptoms like frequent formation of pus pockets in skin, lungs or joints usually begin during infancy. These pus pockets are formed due to infection by staphylococcal bacteria. Treatment usually consists of antibiotics to fight infection and anti histamines to relieve the symptoms like rash and itching. Sometimes drugs can also be administered to modify immune system.
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Answer:
The evolutionary success of this group results from its 1- small size, 2- simplicity and 3- fast reproduction rate
Explanation:
Bacteria are evolutionarily successful organisms that makeup about 15 percent of all biomass on Earth, and they occupy almost all habitats in nature. The small size of these unicellular organisms facilitates the exchange of nutrients and waste products with the surrounding environment. In addition, the relative simplicity of bacteria also facilitates the assembly of cell structures as well as a fast reproduction rate by the fast division of single cells. All these features are critical in order to understand the rapid evolution rate observed in this group.
Disruption of axonal transport would be a good target for his drug.
<h3>What is disruption of axonal transport?</h3>
- Axonal transport defects are among the early molecular events leading to neurodegeneration in mouse models of amyotrophic lateral sclerosis (ALS).
- Gene expression profiles indicate that dynactin-1 mRNA is downregulated in degenerating spinal motor neurons of autopsied patients with sporadic ALS.
- Disruption of axonal transport also underlies the pathogenesis of spinal muscular atrophy and hereditary spastic paraplegias.
- The neurotoxin β,β′-iminodipropionitrile (IDPN) selectively disrupts slow axonal transport without affecting fast anterograde or retrograde axonal transport.
- Impairment of slow axonal transport causes a proximal accumulation of neurofilaments.
- Because neurofilaments regulate axonal diameter, this accumulation leads to a marked swelling of the axon.
- Acrylamide causes decreased axonal transport, also causing proximal accumulations of neurofilaments and swelling.
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