3 is the correct answer. Let me know if you want me to explain why that is the answer.
The properties of filtrate in the glomerular capsule are normally very similar to that of blood plasma except for the lack of Proteins.
<h3>Properties of filtrate in the glomerular capsule</h3>
- The kidneys filter 1100–1200 cc of blood every minute on average. Three layers of blood are filtered as a result of glomerular blood pressure.
- Bowman's capsule epithelium, glomerular blood vessel endothelium, and the basement membrane between these three layers.
- The filtration membrane is composed of these three entities.
- These membranes filter the blood so thoroughly that all but a small portion of the plasma's components enter the bowman's capsule's lumen.
- Additionally, both of them contain diverse electrolytes like sodium, potassium, chloride, and bicarbonate ions as well as glucose, creatinine, urea, and uric acid except for proteins. This procedure is hence termed ultrafiltration.
Therefore, "Proteins" is the right response.
To learn more about the Glomerulus refer to:
brainly.com/question/12993720
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So, you have a type A, which means one has A, you have a B, which means one has B
So right now, Parent A has a blood type of A, and Parent B has a blood type of B
Now a child has AB, which is a possibility figuring that A and B are options
Now that a child has O, which is only a possibility if both parents have O, the Genotype of your parents are AO and BO.
Hope this helped :)
<h2>GPCRs and RTKs</h2>
Explanation:
1) Phosphorylation of receptor can terminate signal or desensitize receptor: GPCRs because when signal persists a protein kinase receptor is activated,this further catalyse the phosphorylation of several serine residue at cytosolic phase of 7 transmembrane
Phosphorylated transmembrane recognized by arrestin protein blocks interaction between trimeric G protein and receptor thus blocks signal transduction
2) Autophosphorylation of receptor can initiate signal: RTKs because receptor dimerization activates intrinsic tyrosine kinase activity by which they phosphorylate its own tyrosine residue on cytosolic phase
3) Structure contains seven transmembrane helices: GPCRs because it consists of single polypeptide and spans the membrane 7 times hence called 7 transmembrane;it recognizes external signal and activates trimeric G protein
4) Ligand binding induces conformational change in receptor: RTKs because normally RTK exist in monomeric form in absence of any ligand molecule but when any ligand molecule binds receptor starts to dimerize
5) Receptor activation causes phosphorylation of its cytosolic subunits: RTKs because when receptor gets activates the intrinsic tyrosine kinase activity by which they phosphorylate its cytosolic subunits
6) An example is the insulin receptor: Insulin receptor is present on plasma membrane and belongs to RTK family and always exist in dimeric form
7) Transports some ligands through the membrane: Neither GPCR nor RTK;ligand gets transported through the membrane through ligand gated channel
8) An example is the epinephrine receptor: Epinephrine is an endocrine hormone produced by adrenal glands in stress condition and prepare body for fight and flight;Epinephrine uses GPCR signalling pathway by four different types of membrane receptors in different tissues
9) Activate heterotrimeric G proteins directly: Transmembrane protein of GPCR recognizes the external signal and activates G protein,activated G protein binds with effector enzyme and activate it which further produce or destroy secondary messenger that carry message from cell surface to cell interior