Answer:
the materials to make the product
The purine or pyrimidine base was removed from the labeled nucleotide base.
A DNA polymerase enzyme recognizes this and replaces the is inserted nucleotide allowing replication to continue. Calibration corrects approximately 99% of these types of errors but is still not sufficient for normal cellular function. Topoisomerases act in the region before the replication fork and prevent supercoiling.
Primase synthesizes RNA primers complementary to the DNA strand. Cells have various mechanisms to prevent mutations and permanent changes in DNA sequences. During DNA synthesis, most DNA polymerases check their work by repairing most mismatched bases in a process called proofreading. Errors can occur during the process of DNA replication. Nucleotide bases can be is inserted, deleted, or mismatched in a DNA strand.
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The presentation of an 18-month-old with hereditary fructose intolerance (HFI), deficiency of aldolase B and low blood glucose levels is due to accumulation of fructose-1 phosphate and the inhibition of glycolytic-gluconeogenic pathways.
Explanation:
Hereditary fructose intolerance leads to deficiency of fructose-1-phosphate aldolase enzyme. This enzyme deficiency leads to accumulation of fructose-1 phosphate in the liver. Fructose-1 phosphate inhibits the action of phosphorylase enzyme which monitors the glycogen to glucose conversion. Since glucose formation is reduced, hypoglycemia and lactic acidosis takes place accounting for the patient’s low blood sugar.
DNA Polymerase is the enzyme that joins individual nucleotides!
If a scientist observes an enzyme polypeptide chain arranged in spiral turns that rise upward and are held in place by hydrogen bonds he/she is looking at a tertiary structural arrangement.
