I suggest to drink energy drinks or coffee and stay active while doing your work!
C.) no impact on body fat percentage
C. Billing data! Hope this helps you
In confirming a diagnosis of cirrhosis, a disease which involves hepatocellular (liver cell) death replaced by scar tissue, the only true definitive diagnostic procedure is a liver biopsy.
However, when an invasive procedure as such is contraindicated, liver enzyme levels (LFTs) may be tested for in blood serum to estimate overall function (or lack thereof in vast cell death throughout the organ as seen in cirrhosis) of hepatocytes (liver cells). These enzymes include: alanine aminotransferase (ALT), aspartate aminotransferase (AST), and often gamma-glutamyl transferase (GGT). Also an abdominal ultrasound (US) of the right upper quadrant can determine the consistency of the liver, i.e. density of sanguinous living tissue versus dead scar tissue. Together, the serum enzyme levels and an US showing widespread hepatic scar tissue (indicative of cirrhosis) are enough to point to cirrhosis. However, true definitive evidence, as aforementioned, is only confirmed via US- or CT-guided tissue biopsy.
*note: CT stands for Computed Tomography, as in the old term "CAT-scan"
The next recommended intervention is the adenosine 6mg IV push. Adenosine is the main drug used in the action of steady narrow complex SVT or as called as supraventricular Tachycardia. It can now also be used for even monomorphic wide complex tachycardia. When given as a rapid IV bolus, adenosine slows cardiac transmission affecting conduction through the AV node. The quick bolus of adenosine also disturbs return SVT initiating the pathways over the AV node and restores sinus rhythm in patients with SVT.The initial dose of adenosine should be 6 mg accomplished fast over 1-3 seconds surveyed by a 20 ml NS bolus. If the patient’s beat does not change out of SVT within 1 to 2 minutes, a second 12 mg dose may be given in similar fashion. Determinations must be finished to manage adenosine as rapidly as likely. A lesser primary dose of 3mg should be used for patients captivating dipyridamole or carbamazepine as these two prescriptions potentiate the effects of adenosine. Also, lengthy asystole has stood become with the use of normal doses of adenosine in heart transfer patients and central line use. Consequently, the lower dose 3mg may be measured for patients with a central venous line or a history of heart transfer.