Answer:
a. Amyloid fibrils have a high amount of β-sheet structure.
c. Because most newly synthesized proteins fold correctly, the accumulation of misfolded proteins (or fragments) tends to occur slowly, thus explaining the slow onset of disease.
e. Proteins that form amyloid fibrils are normally soluble.
Explanation:
Amyloids can be defined as fibrillar protein aggregates composed of a β-sheet secondary structure. In general, the amyloid fibrils are composed by soluble proteins that are assembled to form insoluble resistant to degradation fibers. In humans, defective amyloid proteins produced by misfolding are known to produce diseases (amyloidosis) capable of altering the function of tissues and organs. Systemic forms of amyloidosis are common in neurodegenerative disorders.
Agonist/antagonist
Agonist are drugs that bind and activate a receptor with effecting the response.If the effect results in maximum response it is considered as full agonist. However if the drug that binds to the receptor results only into partial efficacy in relation to the full activation of the receptor, this is considered as partial agonist.
Antagonist are drugs that blocks a biologic response of a hormone or another drug. They usually have affinity to the receptors but has no efficacy. This drug will just inhibit the activation of the receptors by the aganist.
Answer:
C
Explanation:
Dead zones are are areas of low oxygen or hypoxia. They are usually caused by nutrient pollution that leads to algae blooms, but there are other factors that contribute
Hope this helps :)
<span>A plant's plastids contain its chlorophyll, which enables it to photosynthesize. If it lost the plastids, it would not be able to photosynthesize, which is the only way it can get food, so it would die.</span>
Answer:
Above
Explanation:
The anthers are always above the stigma.
