The "thing that hangs in the back of your throat is called a UVULA, its main function is to prevent food from entering your nasal cavity
Since Mary has a Type A blood, she is either AA or AO while John is either BB or BO. Since both John's parents are Type AB, he can only have the either A or B as an allele and not O so he is BB. Since John can only give the B allele, all of his children must have the B allele in their blood types. So the adopted one is the child with type A blood.
The correct answer is D. Precambrian Time - Paleozoic Era - Mesozoic Era - Cenozoic Era. This is the correct order of the geologic time scale, from oldest to most recent. Thank you for posting your question. I hope this answer helped you. Let me know if you need more help.
Answer:
True
Explanation:
Two parents with recessive traits (a trait that is not on display) would create offspring with one dominant trait.
Long-term potentiation (LTP) is considered a cellular correlate of learning and memory. The presence of G protein-activated inwardly rectifying K(+) (GIRK) channels near excitatory synapses on dendritic spines suggests their possible involvement in synaptic plasticity. However, whether activity-dependent regulation of channels affects excitatory synaptic plasticity is unknown. In a companion article we have reported activity-dependent regulation of GIRK channel density in cultured hippocampal neurons that requires activity oF receptors (NMDAR) and protein phosphatase-1 (PP1) and takes place within 15 min. In this study, we performed whole-cell recordings of cultured hippocampal neurons and found that NMDAR activation increases basal GIRK current and GIRK channel activation mediated by adenosine A(1) receptors, but not GABA(B) receptors. Given the similar involvement of NMDARs, adenosine receptors, and PP1 in depotentiation of LTP caused by low-frequency stimulation that immediately follows LTP-inducing high-frequency stimulation, we wondered whether NMDAR-induced increase in GIRK channel surface density and current may contribute to the molecular mechanisms underlying this specific depotentiation. Remarkably, GIRK2 null mutation or GIRK channel blockade abolishes depotentiation of LTP, demonstrating that GIRK channels are critical for depotentiation, one form of excitatory synaptic plasticity.
Learn more about receptors here:
brainly.com/question/11985070
#SPJ4
