Answer:
True.
Explanation:
The immunoglobulins, MHC ( major histocompatibity complex) and TCR (T cell receptor) plays an important role in the immune system. They protect the individual from the harmful antigens.
The evolution of immune system has done the duplication process. The genes gets duplicated and evolved to perform the specific function. The Immunoglobulin, TCR, and MHC gene evolve by the process of duplication. These genes mainly acts against the specific pathogen and shows the selection according to the primordial antigen receptors.
Thus, the answer is true.
I assume that the mushroom gets eaten by an animal. If that's not the answer you're looking for.. my apologies.
???? What’s the 3, 4, 5, and 6 for did u forget to put the numbers
Answer:
Azide and Carbon monoxide block electron transport and proton pumping via inhibition of Complex IV.
Rotenone blocks electron transport and proton pumping via inhibition of Complex I.
Atractyloside blocks electron transport and ATP synthesis by inhibiting the exchange of ATP and ADP across the inner mitochondrial membrane.
Explanation:
The mitochondrial electron transport chain is composed of different complexes integrated into the inner mitochondrial membrane, which function to generate an electrochemical proton (H+ ions) gradient that is then used to drive the synthesis of ATP, the energy coin of the cell. The mitochondrial complex I catalyzes the electron transfer from nicotinamide adenine dinucleotide (NADH) to ubiquinone and then transports H+ ions across the inner mitochondrial membrane. Moreover, the mitochondrial complex IV, also known as Cytochrome c oxidase, receives electrons via the cytochrome pathway in order to complete the reduction of oxygen (O2) into two molecules of water (H2O). Azide is a selective inhibitor of the complex IV which acts as an uncompetitive inhibitor with regards to O2, while carbon monoxide (CO) binds to the cytochrome c oxidase enzyme, thereby suppressing its activity and thus disturbing ATP synthesis. Rotenone is a potent inhibitor of the mitochondrial complex I that inhibits electron transfer from the iron-sulfur centers in the complex I to ubiquinone, thereby blocking ATP synthesis. Finally, atractyloside is a competitive inhibitor that inhibits the transport of ADP across the mitochondrial inner membrane by blocking the ADP/ATP translocator, thereby preventing ADP from entering mitochondria.