Answer: The cell membrane invaginates and pinches off, creating a vesicle within the cell.
Explanation:
Endocytosis is an active transport mechanism, for particules to be transported, the plasma membrane of the cell invaginates, forming a pocket around the target particle. The pocket then pinches off, resulting in the particle being contained in a newly created intracellular vesicle formed from the plasma membrane.
Answer:Dispersal
Explanation: This refers to offspring moving away from their parents. This prevents the offspring from competing with the parents for resources such as light or water. For example, dandelion seeds have “parachutes.” They allow the wind to carry the seeds far from the parents
Answer:
B. Premature stop codon mutation
Explanation:
Yes. True vertebrates have a true head that develops from a neural crest of cells and hard structures surrounding the notochord.
The neural crest, which forms early in the development process in vertebrate embryos, is a fold on the neural plate where the neural and epidermal ectoderms converge. As an embryo grows, the neural crest produces neural crest cells (NCCs), which can differentiate into a variety of different cell types and contribute to tissues and organs.
The notochord is a temporary structure that plays a crucial role in higher animals. It secretes substances that communicate with all neighbouring tissues, telling them where they are and what will happen to them.
Therefore, True vertebrates have a true head that develops from a neural crest of cells and hard structures surrounding the notochord.
Learn more about notochord here:
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The scientist should notice that the cancer cells have a <u>higher</u> rate of cell division and have a <u>lower</u> response to inhibiting factors. Cell groups that are cancerous should have a <u>lower</u> percent of cells in interphase than noncancerous cells.
Explanation:
The general characteristics of cancerous cells are defined by the ‘hallmarks of cancer’ like ‘enable replicative immortality, activate invasion and metastasis, resist cell death, evade growth suppressors’ etc. All these facts prove that they grow and divide at a higher rate and undergo uncontrollable mitotic cell division leading to proliferative invasion.
The cancerous cells also show low or no response to inhibiting factors like growth suppressors like p53, cyclins and other anti-tumor drugs.
The number of cancer cells in interphase is lower than noncancerous cells because interphase is a phase of cell growth, development and differentiation. Cancer cells undergo uncontrollable mitosis and more time is spent after the interphase to facilitate rapid cell division rather than growth or development.
