Answer:
This question lacks options; the options are:
A) Deletion mutation
B) Missense mutation
C) Inversion mutation
D) Nonsense mutation
The answer is D) Nonsense mutation
Explanation:
A mutation in genetics refers to the change in the nucleotide sequence of a gene or DNA segment. The occurrence of mutation in a gene leads to different effects, which is the basis of the different types of mutation. The type of mutation described in this question is called a NONSENSE MUTATION, which is a type of base substitution mutation.
Nonsense mutation is the mutation in which one nucleotide base in the sequence gets replaced by a STOP CODON (UAA, UAG, UGA), leading to the truncation of the amino acid sequence and hence, the production of a non-functional protein.
This is the case in this mutated gene sequence whose normal sequence codes for: Pro-Met-Glu-Arg-Asp. The mutation causes the codon specifying Glutamine to be replaced by either of the STOP codons, thereby, stopping the translation process. The stop to the translation process causes the amino acid sequence to be truncated and a non-functional amino acid sequence is formed: Pro-Met-
Use the following terms to describe in detail how you visually perceive an object that you can see right now.
Answer: To see for example an apple first light that passes through the pupil is eventually reflected on the pupil, activating neurons in the retina. light waves that were reflected off the apple are changed into neural impulses (transduction) at the point of the retina, where neurons fire in response to light waves. Action potentials are released when neurons fire, sending an electrical charge through the neuron. Excitatory neurotransmitters are released when the action potential reaches the axon terminal, and that these neurotransmitters increase the chances that the next neuron will fire. Of course the neural message from the retina first passes through the thalamus, and then the thalamus routes the impulse elsewhere in the brain.
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Cellulose makes the trunk of a tree sturdy.
The antibodies have epitopes that bind to the paratropes present on the antigens. When an antibody is made specifically to target a particular kind of antigen (say A), its epitope will be a structural compliment to the paratrope. When an antibody encounters two different antigens (say A and B) with structurally similar paratropes, it will bind specifically to A and non-specifically to B, as the antibody is not made in response to antigen B. This non-specific binding is referred to as blocking.
