75% species go extinct to be considered a mass extinction
Glucose turns into ATP or ENERGY during the process of cellular respiration ..
<span>The glucose is broken down into 2 molecules of pyruvate, which are two smaller molecules. A net yeild of 2 ATP and 2 NADH result. Each pyruvate is connected to a coenzyme. The resulting molecule is called Acetyl CoA. That reaction also gives off 2 molecules of C02. The Acetyl CoA enters the Krebs Cycle, from which (through a series of steps), 2 more ATP, 6 NADH, 2 FADH2, and 6 CO2 are formed. The 6 NADH and FADH2 (which are coenzymes) move on to the electron transfer chain. Here, they give up their H+ and electrons to the chain. The electrons reduced the proteins on the chain, allowing H+ from outside the cell to be brought in. Bringing this H+ into the cell builds up the concentration. When the concentration gets high enough, the H+ wants to go back out of the cell. The only way to do this is through the ATP synthase. When is passes through this, the synthase combines an ADP with an inorganic phosphate, forming ATP. The typical yeild is 32 ATP from this, giving a total of 36 when you add in the ATP from glycolysis and the Krebs cycle.</span>
These proteins allow substances to enter that wouldn't be able to otherwise. Only small hydrophobic substances can enter cells. However, these proteins aid crucial substsances in entering the cell.<span />
Answer: flexor, hamstring muscle
Explanation:
Answer:
Suppression of Abl reduces the severity of abnormal axonal guidance in PTP mutants
Explanation:
Axon guidance is a neural development subfield relating to the mechanism by which neurons send out axons to meet their right targets.
ABL gene is a protooncogene that encodes a protein tyrosine kinase involved in a variety of cellular processes. PTPs are known to signal a number of cellular processes, including cell development, differentiation, mitotic cycle, and oncogenic transformation.
Suppression of Abl reduces the severity of abnormal axonal guidance in PTP mutants.
