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Jobisdone [24]
3 years ago
15

Which of the following will lead to speciation? Select one of the options below as your answer. A. Variation in gene pool B. Lac

k of mutations C. Absence of natural selection.
Biology
2 answers:
antoniya [11.8K]3 years ago
7 0
<span>The answer is A. Variation in gene pool
Speciation is a formation of a new species during the evolution line. When there is a variation in a gene pool, a unique combination between genes could be formed during the mating process. This will create an offspring with unique gene pool that will lead to speciation</span>
icang [17]3 years ago
5 0
Answer is a. variation in gene pool
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Is one side of DNA is TCA, what is the other side?<br><br> A. AAA<br> B. TCA<br> C. AGT<br> F. TGA
tensa zangetsu [6.8K]

the answer is c: "AGT " BECAUSE A PAIRS WITH T AND C PAIRS WITH G

8 0
2 years ago
A mutation creates a dominant negative allele of a particular gene. The normal allele of the gene encodes a protein that forms a
Wewaii [24]

Answer:

87.5% of the trimers in the cell will be inactive.

Explanation:

A monomer is a single subunit. Look at an easy comparison. If a monomer is a ball. For the protein to be functional, it needs to be a trimer .A trimer = 3 monomers joined / bound together with bonds. Therefore, if a monomer is a ball, we need 3 balls, we attach it together and that is the primer . if one ball becomes missing, that protein won't work .

In chemistry, a trimer is regarded as three molecules or ions of the same substance (that is, same type) which are associated together .

Double negative : Each gene has 2 alleles. So, for a positive protein, both allele needs to be same, let's say + means on. So for a x protein , if it's x+/x+ it means the protein is homozygous and active. If for some mutation, it's x+/x- , it's a heterozygous protein. Double negative means when it is x-/x- and this means the protein has lost is function . It's not functional at all.

The heterozygous cell will form one or more subunits has the mutant structure when the gene is expressed. From the question if there is a mutation in dominant negative allele, it will cause the inactivation of one trimer resulting in 87.5% inactive trimers for a heterozygous individual.

There are 87.5 % percent of the trimers present in the cell which will be inactive for a heterozygous individual, If at least one of the subunits has the mutant structure.

The probability for each individual subunit to be a mutant is 1/2

Gene codes for a protein that forms trimer so the percentage of the trimers present in the heterozygous individual is third power of 1/2 =1/8

1/8=0.125 will be active.

1- 0.125 will be inactive.

=0.875 will be inactive.

0.875 × 100= 87.5%

Therefore 87.5% will be inactive.

8 0
3 years ago
A major difference between mammalian spermatogenesis and oogenesis is _____.
Brut [27]
A major difference between mammalian spermatogenesis and oogenesis is <span>the number of gametes made per month.</span>
3 0
2 years ago
Mention any two similarities between simple perment and complex permanent tissue
enyata [817]

Both types of tissues are made of differentiated cells that have a specific function.

Both types of tissues contain cells that lost the ability to divide

4 0
3 years ago
DCCD (dicyclohexylcarbodiimide) inhibits oxidative phosphorylation when the substrate is mitochondrial NADH. DCCD is a drug that
VARVARA [1.3K]

Answer:

A) Decreases cellular energy production

B) DCCD also affects K+ transport

Explanation:

A) Consequences are of DCCD on cellular energy production: <em>Decreases cellular energy production</em>

ATP-synthase pump is composed of two subunits: F1 catalytic subunit that synthesizes ATP, and F0 proton pumping subunits, that transport H+ through the membrane. F1 subunit might act independently of F0 to produce ATP, but this molecule can not be released without H+ gradient, which generates a movement necessary for ATP release from the catalytic center.

When any of the parts composing F0 react with DCCD, the subunit can not transport H+ through the membrane. DCCD inhibits the enzyme activity by blocking the protons´ flow.

As DCCD blocks the protons´ flow, and the protons´ flow is necessary to release the ATP molecule from the F1 subunit, no other ADP + Pi can enter to F1 subunit, and the production of ATP stops.

B) Other cellular effects of DCCD

There seem to be other effects of DCCD on cell activity, some of which are still under study. To name a few:

  • Diimide from DCCD seems to stimulate cytochrome b reduction and inhibits its reoxidation by ferricyanide.
  • When exposing the cell to high concentrations of DCCD for a long time, might occur an alteration in the electron transporting chain
  • Inhibition of ubiquinol-cytochrome c reductase activity when exposing the cell to high concentrations of DCCD.
  • Inhibition of K+ transport, associated with the inhibition of H+ transport.

Concerning the effect of DCCD on the K+ transport, DCCD stops the extrusion of H+ and the consequent intrusion of K+.

DCCD strongly inhibits the simultaneous flow of H+ and K+. First, it inhibits H+ flow, acidification of the environment stops, but at this point, K+ keeps moving through the membrane. Once the H+ flow has ceased, the K+ flow slowly decreases until it finally stops moving. There is a lag time in the DCCD effect on K+ flow to the instantaneous effect on H+ flow.

5 0
3 years ago
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