During meiosis but not during mitosis, which of the following is true? __.
A haploid gametes are produced that are identical in their allelic composition
B the cytoplasm divides
C chromosomes line up in the center of the cell during metaphase
D genetic variation among the daughter cells is increased
E two identical daughter cells are produced
Answer:
D. genetic variation among the daughter cells is increased
Explanation:
Meiosis is a process where a single cell divides twice to produce four cells containing half the original amount of genetic information. Meiosis occurs in germ cells, as a result; genetic variation among the daughter cells is increased. These cells (germ cells) are our sex cells – sperm in males, eggs in females.
In meiosis, the haploid gametes that are produced are not identical in their allelic composition.
In prophase 1, the chromosome pair up.
During metaphase, The chromosome pairs line up next to each other along the center (equator) of the cell.
Answer:
It was a multi celled organism
Explanation:
Answer:
The given blank can be filled with location of gene expression.
Explanation:
A regulatory sequence refers to a section of a molecule of nucleic acid that possesses the tendency of declining or upsurging the expression of particular genes within an organism. The regulation of gene expression is an important characteristic of all living species and viruses.
In the given case, it is essential to use the regulatory sequence of a milk gene when developing a recombinant gene as a regulatory sequence monitors the location of gene expression.
I had this question I think it’s gamma rays
Answer:
One of the common genetic disorders is sickle cell anemia, in which 2 recessive alleles must meet to allow for destruction and alteration in the morphology of red blood cells. This usually leads to loss of proper binding of oxygen to hemoglobin and curved, sickle-shaped erythrocytes. The mutation causing this disease occurs in the 6th codon of the HBB gene encoding the hemoglobin subunit β (β-globin), a protein, serving as an integral part of the adult hemoglobin A (HbA), which is a heterotetramer of 2 α chains and 2 β chains that is responsible for binding to the oxygen in the blood. This mutation changes a charged glutamic acid to a hydrophobic valine residue and disrupts the tertiary structure and stability of the hemoglobin molecule. Since in the field of protein intrinsic disorder, charged and polar residues are typically considered as disorder promoting, in opposite to the order-promoting non-polar hydrophobic residues, in this study we attempted to answer a question if intrinsic disorder might have a role in the pathogenesis of sickle cell anemia. To this end, several disorder predictors were utilized to evaluate the presence of intrinsically disordered regions in all subunits of human hemoglobin: α, β, δ, ε, ζ, γ1, and γ2. Then, structural analysis was completed by using the SWISS-MODEL Repository to visualize the outputs of the disorder predictors. Finally, Uniprot STRING and D2P2 were used to determine biochemical interactome and protein partners for each hemoglobin subunit along with analyzing their posttranslational modifications. All these properties were used to determine any differences between the 6 different types of subunits of hemoglobin and to correlate the mutation leading to sickle cell anemia with intrinsic disorder propensity.
Explanation:
