The base analog 5-bromouracil can tautomerize. one tautomer behaves like thymine and the other behaves like cytosine. if 5-bromo
uracil is used as a mutagen, what is the minimum number of times replication must occur to mutate the codon for proline (ccc) into the codon for serine (tcc)? multiple choice three generations 5-bromouracil cannot induce this mutation one generation two generations
5-Bromouracil has three tautomers that have different matching properties. The keto form is complementary to adenine, while one of the enol forms is complementary to guanine.
As a result, 5-bromouracil is likely to pair with both adenine and guanine depending on its tautomeric state at the time of replication, which introduces mutations as the replications. During the first replication the analogue will be incorporated in the DNA (in front of a T for example). At the second replication (second generation), the analogue will have a new base in front of it in function of its tautomic form (ie instead of having in front of a T it will have a C and a this moment the mutation becomes complete).
In eukariotes, cells that have a neculeus, the dna is found in the neculeus, not the cytoplasim so that is false... I dunno if that is what u were asking...
Adding large amounts of salt into your body causes your cells to become hypoglycemic (net movement into the cell outweighs net movement out) and causes the cells to boost. Via negative feedback loops, your body feels thirsty to dilute the salt in your cells to return them back to normal concentrations.
If you have one sickle cell gene, you have sickle cell trait and you will not develop the sickle cell disease. If you inherit two sickle cell genes, you have sickle cell anemia.
