The difference in concentration between solutions on either side of a cell membrane is a concentration gradient.
In the field of biology, a concentration gradient can be described as a difference in the concentration of molecules inside and outside of a cell. It is due to concentration gradient that molecules move into and out of a cell through the cell membrane.
Some molecules move from an area of higher concentration gradient to an area of lower concentration along the concentration gradient. Diffusion is an example of such a process.
On the other hand, some molecules move from an area of lower concentration to an area of higher concentration against the concentration gradient. Active transport is an example of such a process.
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<h2>Answer:</h2>
The correct options are 1,4 and 5. By looking at phylogenetic tree we can conclude this;
1. Species A and C may have shared features from a common ancestor.
4. Species B and E may have shared features from a common ancestor.
5. Species A is more related to species B than species B is related to species C
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Long-term potentiation (LTP) is considered a cellular correlate of learning and memory. The presence of G protein-activated inwardly rectifying K(+) (GIRK) channels near excitatory synapses on dendritic spines suggests their possible involvement in synaptic plasticity. However, whether activity-dependent regulation of channels affects excitatory synaptic plasticity is unknown. In a companion article we have reported activity-dependent regulation of GIRK channel density in cultured hippocampal neurons that requires activity oF receptors (NMDAR) and protein phosphatase-1 (PP1) and takes place within 15 min. In this study, we performed whole-cell recordings of cultured hippocampal neurons and found that NMDAR activation increases basal GIRK current and GIRK channel activation mediated by adenosine A(1) receptors, but not GABA(B) receptors. Given the similar involvement of NMDARs, adenosine receptors, and PP1 in depotentiation of LTP caused by low-frequency stimulation that immediately follows LTP-inducing high-frequency stimulation, we wondered whether NMDAR-induced increase in GIRK channel surface density and current may contribute to the molecular mechanisms underlying this specific depotentiation. Remarkably, GIRK2 null mutation or GIRK channel blockade abolishes depotentiation of LTP, demonstrating that GIRK channels are critical for depotentiation, one form of excitatory synaptic plasticity.
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It allows it to hide under but still be able to see above it to catch flies
Answer:
Label A is the amino group
Label B is the R group
Label C is the carboxyl group
Explanation:
