Answer:
Immediately following the arrival of the stimulus at the axon terminal there is a short period called the latent period.
Explanation:
When a stimulus reaches the terminal of an axon, a period known as the latent period begins. This period has a short duration and during it it is possible to observe the blockage of new stimuli in the nerve fiber. Thus, the new stimuli fail to trigger an impulse and no muscle tension to be observed.
While the latent period is not finished, the neurotransmitter is released through the action of exocytosis that moves until it gets access to the receptor capable of activating the muscle cell.
You subtract the number of protons and the mass to get the number of neutrons.
Numerous degenerative neurological conditions, most notably Parkinson's disease, have been linked to an excessive buildup of alpha synuclein (a-syn) in the brain. Intraneuronal inclusions, often known as Lewy bodies, are neuropathological characteristics seen in Parkinson's disease, Lewy body dementia, and other synucleopathies. The aggregation of a-syn is their main structural component. A-syn accumulation, aggregation, and ensuing Lewy body formation can be attributed to a variety of biological processes. These include genetic changes in parkin, synuclein, or the deubiquitinating enzyme ubiquitin C-terminal hydrolase (UCH-L1), which results in less efficient removal of a-syn via the ubiquitin proteasomal pathway (UPP). Additionally, environmental variables and an age-related decline in antioxidant defense mechanisms that heighten oxidative stress and can have an impact on the formation or clearance of a-syn are intracellular insults.
We focused on changes in the aggregation and clearance of a-syn as impacted by the UPP and the oxidative stress pathways in our dynamic models of a-syn processing in both normal and various disease states. A free radical profile similar to that observed in vivo after exposure to the neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine is produced during simulation of enhanced oxidative stress (MPTP). To replicate the kinetics of a-syn that correlates to the neuropathology reported for the sporadic and hereditary types of Parkinson's disease, different model parameters of oxidative stress, UPP failure, or both routes are used. With the use of this in silico model, it is possible to evaluate the kinetics of pathway elements and more accurately identify and validate key pharmaceutical targets.
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Explanation:
the light energy is converted to heat energy in order to break the covalent bonds in the water molecules that are combined with CO2 to create glucose
This is because the water is dew.
Maybe we should probably check how that what gets to the ground in the morning. At the point when the surface of a question winds up plainly colder because of sending its idle warmth out, the current dampness in the air contracts among itself.
