<span>The birds with the longer beaks will be fittest in the environment post-drought because they will be able to gather resources (food) hence survive. This will enable these bords to reach reproductive again and pass down the genes. It is important to note that fitness in nature is determined by the capacity to reproduce.
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<span>Natural selection will change the allelic frequency such that the allele for long neck will be more in the population while that of short beaks will reduce. This is because short beaks trait will not be able to reach reproductive age (due to starvation) because of the incapacity to gather food <span>resource hence their genes will keep reducing in the subsequent generations.
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<span>Natural selection is a natural selective force that acts on a population and results to variation (differential) in the survival of the individuals in the population due to variations in phenotypes in the population. The most successful traits (phenotype that is best suited for the environment) are able to reach reproductive age, find a mate and reproduce hence leave its genes in subsequent generations</span>
Smallpox, herpes, rabies, and the common cold :)
Glaucoma - a disease that damages the optic nerve that sends pictures to the pain. if left untreated, it can lead to loss of peripheral vision.
Explanation:
Γ-Aminobutyric acid (GABA) is a non-protein amino acid that is widely present in microorganisms, plants and animals. It is the main inhibitory neurotransmitter in the central nervous system (CNS) of mammals.
It plays the leading role in reducing neuronal excitability throughout the nervous system. In humans, GABA is directly responsible for the regulation of muscle tone.
Although, in chemical terms, it is an amino acid, in the scientific and medical communities they rarely refer to GABA as such because the term "amino acid" by convention refers to the α amino acids and GABA is not. It is also not considered as part of any protein.
In spastic diplegia in humans from an early age, GABA absorption is negatively affected by the nerves damaged by the lesion in the upper motor neurons characteristic of the condition which leads to the development of muscular hypertonia signaled by those nerves that are incapable of absorbing GABA.
