Question: Is the pre-synaptic or the post-synaptic neuron damaged? Explain your rationale in detail (in other words, detail how you came to your conclusion). You're going to have to really think on this one. It's not something you can look up in the text. You will need to use what you have learned about the autonomic nervous system and some critical thinking to arrive at the answer.
Horner's Syndrome riddle: Ach is the neurotransmitter at all ANS preganglionic fibers, including SNS. The pt has decreased sweating and pupil constriction, indicating blockage of SNS transmission. When the MD gave ACh it induced sweating, but not pupil dilation. This is because the sweat glands are an exception in normal SNS transmission, as the postganglionic fibers utilize ACh as their neurotransmitters. Other SNS pathways utilize NE as their neurotransmitter. So ACh acted directly on the sweat gland to produce sweating, but could not stimulate the typical postsynaptic neurons to release NE. When the MD gave NE he saw pupil dilation, but not sweating, indicating the NE worked directly at the iris to produce pupil dilation. This indicaates the problem was with the postsganglionic neuron. If the preganglionic neuron were the problem injecting ACh would have solved both problems.
Answer:
As soon as you cut or scrape skin anywhere on your body, special blood cells called platelets spring into action. Platelets stick together like glue at the cut, forming a clot. This clot is like a protective bandage over your cut that keeps more blood and other fluids from flowing out.
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Answer: O made of polÿsaccharides and chitin
Explanation:
Because of transcription. Transcription is the first step, where the RNA is synthesized.
Answer:
The spindle, shown in figure below, consists of fibers made of microtubules. the spindle starts to form during prophase of mitosis kinetochores on the spindle attach to the centromeres of sister chromatids.
Explanation: Chromosomes line up at the metaphase plate under tension from the mitotic spindle.
