Explanation:
cancer cells continue to grow they fail to listen to the signal that tells them to stop
cancer cells do not stick to nearby cells like normal cells they lack stickiness caused by adhesion molecules, can break free ans float to other regions of the body
cancer cells are immortal normal cells have a lifespan cancer cells defy death on the end of the chromosomes is a structure known as telomere. everytime a cell divieds it becomes shorter to a point where the cell dies cancer cells figures out a way to restore their telomeres so they dont shorten as the cells divide and they become immortal
Your answer is Uplifted! Hope this helps!
"Scarus frenatus" I think
An increase in the atmospheric concentrations of greenhouse gases produces a positive climate forcing, or warming effect. ... The warming effect associated with carbon dioxide alone increased by 36 percent
Answer:
C.Never swims fast
Explanation:
RacerX and Speed protein need to bind to each other for the cilia to beat faster which would make the protozoan swim faster. However, Speed needs to be phosphorylated to be able to bind to RacerX. Thus, phosphorylation of Speed is necessary for the fast movement of cilia.
Threonine residue is phosphorylated in Speed protein. It occurs on its side chain through the formation of phosphoester bond. Apart from threonine, phosphorylation can take place on serine and tyrosine side chains again through the formation of phosphoester bond. It can also occur on histidine, lysine and arginine by formation of phosphoramidate bonds.
Since, alanine can not form any of these bonds it can not be phosphorylated. Hence when threonine is substituted with alanine in Speed protein, it can not be phosphorylated. Racer X wont be able to bind to Speed and cilia wont beat faster. As a result the protozoan will never swim fast.
