Answer:
The activation energy must be reached. The catalyst makes lower energy pathways available.
Explanation:
Answer:
Polar bears consume ringed seals (which are tertiary consumers) as well as fish like trout, salmon, and cod (which are secondary consumers).
Explanation:
Answer:
100% merle with long coats.
Explanation:
<em>All the offspring produced from the cross would be merle with long coats.</em>
<u>From the illustration:</u>
Merle gene is represented by the genotype cmcs.
fgf5 gene determines the coat phenotype. Let the gene be represented by the allele A. A determines long coat traits and it is dominant over its alternate form, b, which determines short coat trait.
Now, two merle (cmcs) dogs were mated. One is true-breeding for long coat (AA) while the other is true breeding for short coat (aa).
cmcsAA x cmcsaa
Progeny genotype = cmcsAa
cmcsAa = merle with long coat.
<em>Since </em><em>A</em><em> is dominant over </em><em>a,</em><em> all the progeny from the cross will be merle with long coat.</em>
Unicellular organisms are organisms which contain single cells , they include bacteria, archaea, protozoa, unicellular fungi, unicellular algae among other organisms. Multi cellular organisms are those organisms made of made up of many cells these includes plants and large organisms. Unicellular organisms are believed to have formed from non-living elements approximately 3.5 billion years ago.
Eukaryotic cells have been confronted throughout their evolution with potentially lethal plasma membrane injuries, including those caused by osmotic stress, by infection from bacterial toxins and parasites, and by mechanical and ischemic stress. The wounded cell can survive if a rapid repair response is mounted that restores boundary integrity. Calcium has been identified as the key trigger to activate an effective membrane repair response that utilizes exocytosis and endocytosis to repair a membrane tear, or remove a membrane pore. We here review what is known about the cellular and molecular mechanisms of membrane repair, with particular emphasis on the relevance of repair as it relates to disease pathologies. Collective evidence reveals membrane repair employs primitive yet robust molecular machinery, such as vesicle fusion and contractile rings, processes evolutionarily honed for simplicity and success. Yet to be fully understood is whether core membrane repair machinery exists in all cells, or whether evolutionary adaptation has resulted in multiple compensatory repair pathways that specialize in different tissues and cells within our body.
