Answer:
A superficial injury to the spinocerebellar tracts results in loss of coordinated movements of the trunk and lower extremities and presents with an abnormal clinical sign termed Ataxia.
Explanation:
Lesions of spinocerebellar tracts are ipsilateral. hope this helps you :)
Prader-Willi syndrome (PWS) is a gentic disorder which has an impact on numerous physiological systems. PWS affected individuals (specifically babies) experience delayed growth, significant hypotonia (low muscle tone), and feeding issues. It does effect circadian rhythms in mice models.
SNORD116, often referred to as HBII-85, is a non-coding RNA (ncRNA) molecule that contributes to the alteration of other small nuclear RNAs (snRNAs). Unlike the majority of other snoRNAs, SNORD116 is not significantly complementary to ribosomal RNA and is expressed widely in the brain (but not in PWS patients).
According to the studies, SNORD116 cause sleep defect in patients with Prader-Willi syndrome. Same observation was seen in mouse models too. Paternal expression of SNORD116 is thought to be a potential gene for the sleep disruptions/circadian rhythm’s that the majority of PWS sufferers.
To learn more about circadian rhythm click here
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Answer:
Yes possible
Explanation:
If mother is heterozygous A then her alleles are iA and iO while father is AB so crossing the two we get AO, AA, AB and BO. Hence proved
Answer:
the presence of cell structures in the cell
Explanation:
<em>If you do not know the reasoning behind this, you may want to read the explanation assignment the teacher adds, or do more research, look at notes, or pay attention during class :)</em>
