Multiple myeloma causes thrombocytopenia, which is an increase in the level of platelets in the blood is the answer
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Answer:
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Explanation:
opens up air way makes lungs syronger
The nurse will perform pulse oximetry to monitor the effectiveness of the oxygen therapy ordered for the client.
<h3>What is pulse oximetry?</h3>
The oxygen saturation level of your blood can be measured with a non-invasive procedure called pulse oximetry.
It can quickly identify even minute variations in oxygen levels. These levels demonstrate how well blood transports oxygen to your arms and legs, which are the extremities that are farthest from your heart. It looks like a little clip and is called a pulse oximeter. It fastens to a body component, usually a finger.
Pulse oximetry is helpful for postoperative patients, monitoring individuals at risk for hypoxia, titrating oxygen therapy, and monitoring patients receiving oxygen therapy.
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Hyperglycemia associated with diabetic ketoacidosis is defined as a blood glucose measurement equal to or greater than 250mg/dl
<h3>What is diabetic ketoacidosis?</h3>
Diabetic ketoacidosis is a medical condition that affects people with diabetes. This happen when when the body of a diabetic patient begin to degrade fat at a very high rate that is too fast. The liver breakdown the fat into a fuel called ketones, which make the blood to become acidic.
Therefore, Hyperglycemia associated with diabetic ketoacidosis is defined as a blood glucose measurement equal to or greater than 250mg/dl
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Subsequent INR readings are influenced by the dose, method, and initial INR of vitamin K. For intravenous vitamin K doses of 2 mg or more, INR decrease is comparable. FFP preadministration has no effect on INR readings 48 hours or more after vitamin K administration.
What is Abstract of Vitamin K dosing to reverse warfarin based on INR, route of administration, and home warfarin dose in the acute/critical care setting?
- Commonly, vitamin K is used to reverse the anticoagulant effects of warfarin. The ideal vitamin K dosage and delivery method that does not lengthen bridging therapy are still unclear.
- To ascertain the elements affecting the level and pace of vitamin K-induced INR reversal in the acute/critical care setting.
- 400 patients' charts from between February 2008 and November 2010 who got vitamin K to counteract the effects of warfarin were examined. International normalized ratios (INRs), intravenous or oral vitamin K doses, and whether or not fresh frozen plasma (FFP) was administered were among the information gathered. INRs were measured 12, 24, and 48 hours before vitamin K treatment.
- At baseline, 12 hours, 24 hours, and 48 hours, respectively, intravenous vitamin K decreased INR more quickly than oral vitamin K (5.09, 1.91, 1.54, and 1.41 vs. 5.67, 2.90, 2.14, and 1.58). Subsequent INR values were impacted by baseline INR (p 0.001), method of administration (p 0.001), and vitamin K dosage (p 0.001). For intravenous vitamin K doses of 2 mg or more, there was a similar drop in INR. Home warfarin dose had no effect on INR responses to intravenous or oral vitamin K (p = 0.98 and 0.27, respectively). FFP had no effect on INR readings 48 hours later. Although larger vitamin K doses and longer anticoagulation bridge therapy appeared to be related, neither the incidence (p = 0.63) nor the duration (p = 0.61) were statistically significant.
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