Answer:
AaGLIZZYYYG YES ORRR HAMBURGERR
Answer:
Explanation:
The bone marrow produces stem cells, this is what makes your bodys blood cells – red cells, white cells and platelets. The erythropoietin sends a message to the stem cells telling more of them to develop into red blood cells, rather than white cells or platelets.
The health care provider suspects the child has developed: Acute post infectious glomerulonephritis.
Acute poststreptococcal glomerulonephritis (APSGN) shows symptoms like infection of the skin or throat and caused by nephritogenic strains of group A beta-hemolytic streptococci. Poststreptococcal glomerulonephritis (GN) is a kidney disease that occurs after infection with certain strains of streptococcal bacteria. The kidneys are responsible for removing waste from the body, regulating electrolyte balance and blood pressure, and stimulating the production of red blood cells. A child is recovering from a bout with group A beta-hemolytic Streptococcus infection. The child returns to the clinic a week later complaining of decrease in urine output with puffiness and edema noted in the face and hands. So these are the symptoms of Acute post infectious glomerulonephritis.
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I would say the pineal gland. If there is a reduction in pinealocytes function, melatonin production gets reduced.
The medications would the nurse anticipate administering to this patient include Pyridostigmine, Azathioprine and Prednisone.
Pyridostigmine (Mestinon) is an anticholinesterase that enhances the action of acetylcholine neurotransmitters at the neuromuscular junction.
Azathioprine (Imuran) is an immunosuppressant medication (it has immunomodulatory effects).
Prednisone (Deltasone) is a corticosteroid used to suppress the immune response associated with the symptoms above described.
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