ICD-10-CM in this case represents Z38.00 (single liveborn infant) and Q65.32 (Congenital dislocation of left hip, unilateral).
<h3>What is ICD-10-CM ?</h3>
The International Classification of Diseases, Tenth Revision, Clinical Modification code, commonly known as ICD-10-CM, is a coding methodology to analyze health problems and corresponding procedures to prevent and treat them.
The ICD-10-CM codes are widely used in the USA by medical professionals in clinical settings.
In conclusion, ICD-10-CM in this case represents Z38.00 and Q65.32.
Learn more about ICD-10-CM codes here:
brainly.com/question/9442380
#SPJ1
Answer: Mutations in the GALT, GALK1, and GALE genes cause galactosemia.
Explanation: Galactosemia is a disorder that affects how the body processes a simple sugar called galactose. A small amount of galactose is present in many foods. It is primarily part of a larger sugar called lactose, which is found in all dairy products and many baby formulas. The signs and symptoms of galactosemia result from an inability to use galactose to produce energy.Classic galactosemia, also known as type I, is the most common and most severe form of the condition. If infants with classic galactosemia are not treated promptly with a low-galactose diet, life-threatening complications appear within a few days after birth. Affected infants typically develop feeding difficulties, a lack of energy (lethargy), a failure to gain weight and grow as expected (failure to thrive), yellowing of the skin and whites of the eyes (jaundice), liver damage, and abnormal bleeding. Other serious complications of this condition can include overwhelming bacterial infections (sepsis) and shock. Affected children are also at increased risk of delayed development, clouding of the lens of the eye (cataract), speech difficulties, and intellectual disability. Females with classic galactosemia may develop reproductive problems caused by an early loss of function of the ovaries (premature ovarian insufficiency). Galactosemia type II (also called galactokinase deficiency) and type III (also called galactose epimerase deficiency) cause different patterns of signs and symptoms. Galactosemia type II causes fewer medical problems than the classic type. Affected infants develop cataracts but otherwise experience few long-term complications. The signs and symptoms of galactosemia type III vary from mild to severe and can include cataracts, delayed growth and development, intellectual disability, liver disease, and kidney problems.
Answer:
Tablets have a longer shelf life and come in a variety of forms. They can also accommodate a higher dose of an active ingredient than a capsule. They tend to be slower acting and, in some cases, may disintegrate unevenly in your body. Capsules act quickly and most, if not all, of the drug is absorbed
Explanation:
Introduction :-)
The <em>amniotic fluid</em> is the protective liquid contained by the <em>amniotic sac</em> of a <em>gravid amniote</em>. This <em>fluid</em> serves as a cushion for the growing <em>fetus</em>, but also serves to facilitate the exchange of nutrients, water, and biochemical products between <em>mother</em> and <em>fetus</em>.
Importance :-)
- <em>It helps to protect the fetus from trauma to the maternal abdomen.</em>
- <em>It cushions the umbilical cord from compression between the fetus and uterus.</em>
- <em>It has antibacterial properties that provide some protection from infection.</em>
- <em>It serves as a reservoir of fluid and nutrients for the fetus.</em>
<em></em>
Formation :-)
<em>Amniotic fluid </em>is present from the formation of the <em>gestational sac.</em> <em>Amniotic fluid </em>is in the <em>amniotic sac.</em> It is generated from <em>maternal plasma</em>, and passes through the <em>fetal membranes</em> by <em>osmotic</em> and <em>hydrostatic forces</em>. When <em>fetal kidneys</em> begin to function around week 16, <em>fetal urine</em> also contributes to the <em>fluid.</em> In earlier times, it was believed that the <em>amniotic fluid</em> was composed entirely of <em>fetal urine</em>.
The <em>fluid</em> is absorbed through the <em>fetal tissue</em> and <em>skin</em>. After <em>22 to 25 week </em>of<em> pregnancy</em>, <em>fraternization</em> of an <em>embryo's skin</em> occurs. When this process completes around the <em>25th week</em>, the<em> fluid</em> is primarily absorbed by the <em>fetal gut</em> for the remainder of <em>gestation</em>.
<em>Contents :-)</em>
At first, <em>amniotic fluid</em> is mainly water with <em>electrolytes</em>, but by about the <em>12-14th week</em> the <em>liquid</em> also contains <em>proteins, carbohydrates, lipids and phospholipids, and urea</em>, all of which <em>aid</em> in the growth of the <em>fetus</em>.
Hope its helpful :-)
If so, please mark me as brainlist :-)