Don't eat them? Or before cooking rinse them off before cooking ♀️
The scenario will be Son: 25% colorblind daughter: 25% colorblind son: 25% of carrier daughters with normal vision: 25% normal son
<h3>What is color blindness?</h3>
The condition is frequently inherited. Certain eye diseases and medications are also possible causes. Men are more affected than women.
Color blindness is characterized by the inability to distinguish between red and green shades.
A colorblind man's genotype is XcY, and a heterozygous carrier female's genotype is XcX. A cross between XcY and XcX would result in progeny with the following ratio=
Son: 25% colorblind daughter: 25% colorblind son: 25% of carrier daughters with normal vision: 25% normal son.
Thus, the couple is likely to have a son who is half normal and half affected. Similarly, the couple is likely to have 50% normal daughters and 50% colorblind daughters.
For more details regarding color blindness, visit:
brainly.com/question/25621649
#SPJ1
when a homozygous white rabbit (bb) is crossed with a heterozygous black rabbit (Bb). what proportion of white rabbits would the offspring have?
Answer:
Based on the Mendelian fashion cross,
bb is hetezygote reccessive.(white)
Bb is heterozyote dominant.(black)
Therefore,the the cross to show the proportion is below
Bb,Bb,bb,bb,
2 blacks(hetero zygote blacks) and 2 white.(homo zygote whites)
thus 2 of the offspring will be white, in 50%, of the number of offspring
Explanation:
Answer and Explanation:
The Huntington disease is caused by a mutation in the gene that codifies for the Huntingtin protein (Htt). The mutation produces an altered form of the protein leading to the neuron´s death in certain areas of the brain.
The Huntington disease characterizes for being,
- Hereditary, passing from generation to generation. To express the disease, a person must have been born with an altered gene.
- Autosomal, affecting men and women equally, because the mutated gene is located on an autosomal chromosome.
- Dominant, which means that by getting only one copy of the altered gene coming from any of the parents, the receiving person will express the disease. The mutation in the gene dominates over the normal gene copy.
- Expressed by heterozygosis. Most people affected by the disease are heterozygous, with a normal copy and a mutated copy.
There are just a few cases all around the world (3% approximately) in which the disease is expressed with no family history. The progenitors are not affected by the mutation. These cases are very rare and are called <u>"de-novo" mutations.</u> A new mutation is spontaneously produced and it is not inherited from any of the parentals. It consists of an increase in the number of CAG repetitions. In a normal person, the number of CAG repetitions is less than 35. When there are 40 or more repetitions it occurs the disease. But when there are between 35 and 39 repetitions, the penetrance of the disease is incomplete. This is a "gray zone". Those alleles that fall in the gray zone are unstable and might produce the HD. Individuals with these unstable alleles have a tendency to increase the number of repetitions from generation to generation until the number reaches 40 repetitions and the person expresses the disease. This <u>usually occurs in the paternal germinal line</u>, as it is particularly unstable in sperm and probably meiosis greatly affects their instability, causing an increase in the number of CAG repeats.
Ants and bee's don't have a backbone, instead, in most cases, they have an exoskeleton.
Hope this helped:)
