D. ATP (Adenosine TRIphosphate) has one more phosphate group and is higher in energy than ADP (Adenosine DIphosphate). TRI means three, DI means two, so the names of the molecules tell you there's one more phosphate in ATP than in ADP.
That extra phosphate makes ATP higher in energy (when the cell uses that energy to do something - contract a muscle fiber, for instance - the ATP's extra energy is used and it gets converted to ADP).
Changes in mitosis, crossing over, random fertilization and random arrangement in meiosis.
<u>Explanation:</u>
Genetic diversity of sexually reproducing organisms are controlled by many factors and they are - changes in mitosis, crossing over, random fertilization and random arrangement in meiosis.
During crossing over, the sister chromatids of homologous chromosome crossover and exchange their genes which leads to recombination eventually causing genetic diversity.
Random fertilization of male and female gametes leads to the formation of zygote having the genetic content of both maternal and paternal gametes causing variation.
Environmental factors also causes some change in with time.
Answer:
Eukaryotes:
-Organisms with a well defined nucleus are known as eukaryotes. such cells are called as eukaryotic cells.
- Their nucleus is enclosed within the nuclear membrane.
- They do not posses a mitochondria.
- The cell wall is the outer most layer of a eukaryotic cell (only plant cells have a cell wall)
- The cells are divided by a process called mitosis.
Eg: Plants, Animals.
Prokaryotes:
- Organisms without a well defined nucleus are known as prokaryotes. Such cells are called as Prokaryotic cells.
- They lack nuclear membrane.
- Mitochondria, Golgi bodies, chloroplast and lysosomes are absent.
- The genetic material (DNA) is present on the chromosome.
Eg: Every organism coming under the kingdom monera.
Explanation:
The precursor of a mitochondrial matrix protein is found in a mutation in the tom22 signal receptor.
Tom22 and Tom20 function as N-terminal matrix targeting sequences' outer mitochondrial membrane receptor proteins. The buildup of mitochondrial matrix-targeted proteins in the cytosol due to a malfunctioning Tom22 receptor protein may be followed by cytosolic turnover of these proteins.
The N-terminal sequence, which directs the protein to mitochondria and is proteolytically processed upon import3, is produced in approximately 70% of mitochondrial precursor proteins3. To get to the matrix, they must pass via the TIM23 and TOM complexes on the inner and outer membranes, respectively.
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