A. Plants use capillarity to move water from their roots to their leaves.
Answer:
genomic imprinting
Explanation:
Genomic imprinting is a mechanism for regulating gene expression that allows expression of only one of the parental alleles, although both alleles are functional. Unlike most genes in which expression is biallelic, genes that are subjected to this mechanism (imprinted genes) have monoalelic expression; By definition, in an imprinted loci, only one allele is active (maternal or paternal), and the inactive is epigenetically marked by histonic modification and / or methylation of cytosines.
Genomic imprinting can cause some disturbances, among them Prader-Willi syndrome, which is a genetic disorder that involves a partial deletion of chromosome 15q on the paternal chromosome.
Numerous degenerative neurological conditions, most notably Parkinson's disease, have been linked to an excessive buildup of alpha synuclein (a-syn) in the brain. Intraneuronal inclusions, often known as Lewy bodies, are neuropathological characteristics seen in Parkinson's disease, Lewy body dementia, and other synucleopathies. The aggregation of a-syn is their main structural component. A-syn accumulation, aggregation, and ensuing Lewy body formation can be attributed to a variety of biological processes. These include genetic changes in parkin, synuclein, or the deubiquitinating enzyme ubiquitin C-terminal hydrolase (UCH-L1), which results in less efficient removal of a-syn via the ubiquitin proteasomal pathway (UPP). Additionally, environmental variables and an age-related decline in antioxidant defense mechanisms that heighten oxidative stress and can have an impact on the formation or clearance of a-syn are intracellular insults.
We focused on changes in the aggregation and clearance of a-syn as impacted by the UPP and the oxidative stress pathways in our dynamic models of a-syn processing in both normal and various disease states. A free radical profile similar to that observed in vivo after exposure to the neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine is produced during simulation of enhanced oxidative stress (MPTP). To replicate the kinetics of a-syn that correlates to the neuropathology reported for the sporadic and hereditary types of Parkinson's disease, different model parameters of oxidative stress, UPP failure, or both routes are used. With the use of this in silico model, it is possible to evaluate the kinetics of pathway elements and more accurately identify and validate key pharmaceutical targets.
Learn more about Parkinson's disease brainly.com/question/28169444
#SPJ4
Answer:
The identity of a protein has relation with its amino acid sequence. This sequence is determined by the nucleotide sequence of the DNA of the gene transcribed to RNA and translated into amino acids. Protein identity determines its structure and function. The concentration of a protein refers to the percentage of protein that exists in a given solution. One way to quantify the concentration is with a spectrophotometer calculating the absorbance of the protein.
Genetic information.The main job of DNA is to carry the code for making protein.A gene is a stretch of DNA that can be read by proteins called ribosomes, and copied I to type of nucleic acid called massenger RNA.
