There are six aorganells in an animal cell. The Nucleus, Ribosomes, Endoplasmic reticulum, Golgi apparatus, Chloroplasts and the Mitochondria.
I'm guessing the rocket scenario is the first one and the asteroid one is the last. That being said...
1) The fuel in the rocket provides it with the energy it needs to propel itself into space. The rocket throttles at the bottom, allow it to break free from the earth's gravity by pushing against the earth's greater mass.
2) If you are inside of the bus, in a fixed spot not moving, and the bus is moving. That means you are moving in the same direction as the bus. If it stops, you're still moving at the speed that it was once originally moving at. Obviously, since you weigh less than the bus you are going to fly forwards.
3) There is no gravity or friction in space, so items won't slow down no matter what. Even if an object such as a meteor is flung into space with the tiniest bit of force it will keep flying through space at a consistent speed, since there's nothing to slow it down... unless it hits something else.
4) Looking back at the previous answer, the asteroid is flying through space at a consistent speed (which is really fast). Suddenly it comes in close range to the moon which is bigger in size, and thus has a stronger gravitational pull, and pulls the asteroid into it's field.
Ti plasmid encoded octopine and nopaline catabolism in Agrobac terium Ti plasmid-encoded genes required by the micro organism for opine catabolism.
The occ and noc areas in octopine and nopaline Ti plasmids, respectively, are accountable for the catabolism of octopine and nopaline in Agrobacterium. The functions are activated within the presence of the opines with the aid of OccR and NocR, related regulatory proteins, and the promoters incorporate commonplace collection motifs.
we have investigated Ti plasmid in heterologous interactions among the regulators and the promoters. previous experiments the usage of all possible heterologous combos of opines, regulators, and promoters in vivo had demonstrated that handiest the aggregate of nopalme, NocR, and the occ promoter led to restricted promoter activation. We now display that OccR and NocR bind to the heterologous promoters in vitro and in vivo.
The weak or non-existent promoter activation truly located can be explained by the idea that OccR and NocR use distinct activation mechanisms; we investigated protein-brought about DNA bending due to reports that the two regulators vary in this respect.
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