You would need to administer 2.5ml
The most commonly used formula now is that of Mosteller, published in 1987 in The New England Journal of Medicine. According to Mosteller's "simplified calculation of body-surface area In metric terms" the body surface area = the square root of product of the weight in kg times the height in cm divided by 3600.
Answer:
Most likely he has injured his <u>Calcaneal tendon</u> and the bulge is <u>triceps surae or calf muscle.</u>
Explanation:
The calcaneal tendon is the thickest tendon, which is present at the back of the lower leg and begins at the middle of the calf. It is covered with skin and fascia and connects the bones with the muscles.
The calf muscles or the triceps surae, are the muscles present in the calf of the leg.The calf muscles act via the tendon and cause flexion of the knee and the ankle.
<u>While exercising, an overuse of the calcaneal tendon, can result in pulling of the calf muscles from the Achilles tendon. Thus causing a tear in the calf muscle, which appears like a bulge.</u>
Subsequent INR readings are influenced by the dose, method, and initial INR of vitamin K. For intravenous vitamin K doses of 2 mg or more, INR decrease is comparable. FFP preadministration has no effect on INR readings 48 hours or more after vitamin K administration.
What is Abstract of Vitamin K dosing to reverse warfarin based on INR, route of administration, and home warfarin dose in the acute/critical care setting?
- Commonly, vitamin K is used to reverse the anticoagulant effects of warfarin. The ideal vitamin K dosage and delivery method that does not lengthen bridging therapy are still unclear.
- To ascertain the elements affecting the level and pace of vitamin K-induced INR reversal in the acute/critical care setting.
- 400 patients' charts from between February 2008 and November 2010 who got vitamin K to counteract the effects of warfarin were examined. International normalized ratios (INRs), intravenous or oral vitamin K doses, and whether or not fresh frozen plasma (FFP) was administered were among the information gathered. INRs were measured 12, 24, and 48 hours before vitamin K treatment.
- At baseline, 12 hours, 24 hours, and 48 hours, respectively, intravenous vitamin K decreased INR more quickly than oral vitamin K (5.09, 1.91, 1.54, and 1.41 vs. 5.67, 2.90, 2.14, and 1.58). Subsequent INR values were impacted by baseline INR (p 0.001), method of administration (p 0.001), and vitamin K dosage (p 0.001). For intravenous vitamin K doses of 2 mg or more, there was a similar drop in INR. Home warfarin dose had no effect on INR responses to intravenous or oral vitamin K (p = 0.98 and 0.27, respectively). FFP had no effect on INR readings 48 hours later. Although larger vitamin K doses and longer anticoagulation bridge therapy appeared to be related, neither the incidence (p = 0.63) nor the duration (p = 0.61) were statistically significant.
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