Flu<span>, </span>hepatitis A<span>, </span>hepatitis B<span>, </span>chickenpox<span>, </span>herpes zoster<span> (</span>shingles<span>), </span><span>cancer</span>
Answer:
Lysine
Explanation:
lysine residues on the histone tails of the octamer cn be activated by both acetylation and methylation patterns to influence accessibility or silencing of the genes respectiviely. for example, acetylation of H3K27 (histone 3 lysine residue 27) brings about a region of active chromatin allowing access to transcription activity while its trimethylation will cause silencing of the associated gene at that particular area (no expression of that gene)
My best guess would be C because when you split something up the mix’s of animals decreases and it would be a disturbance
<span>Reactive or evocative correlation. An example would be a child that shows particular interest and talent to sports. The parents are not particularly inclined to sports and grew up not playing them, but provides the child with opportunities to play and practice them. This action by the parents reinforces the child's genetic tendency to sports.</span>
Clinical death is the medical term for cessation of blood circulation and breathing, the two necessary criteria to sustain human and many other organisms' lives.
It occurs when the heart stops beating in a regular rhythm, a condition called cardiac arrest.
Brain injuries start to accumulate almost immediately after Clinical Death.
Full recovery of the brain after more than 3 minutes of clinical death at normal body temperature is rare.
Usually brain damage or later brain death results after longer intervals of clinical death even if the heart is restarted and blood circulation is successfully restored.
Although loss of function is almost immediate, there is no specific duration of clinical death at which the non-functioning brain clearly dies.
The most vulnerable cells in the brain, CA1 neurons of the hippocampus, are fatally injured by as little as 10 minutes without oxygen.
However, the injured cells do not actually die until hours after resuscitation.
Brain failure after clinical death is now known to be due to a complex series of processes called Reperfusion Iinjury that occur after blood circulation has been restored, especially processes that interfere with blood circulation during the recovery period.
Hope this helps!!!
~Alkka♥
