Answer:
The possible fate of the cell that it may turn cancerous.
Explanation:
The cells present in the body generally work in harmony. However, if a cell attains a mutation, it can make it proliferate in the case when it should not do, and make it thrive in the case when other cells are dying. Due to proliferation, the unusual cell produces more abnormal cells also known as cancerous cells. These cancerous cells become more favorable in comparison to the normal cells due to the phenomenon of natural selection. These cells eventually result in a lethal form of tumors.
In the normal cells, the destructed gene or the damaged cells get repaired easily, in case if the damage is worse the cell dies. A protein known as p53 helps in repairing damaged cells or kills them if the damage is too severe. But in the case of cancer cells, the p53 protein does not work appropriately as they possess a mutated or changed form of p53 protein. Thus, in the case of cancerous cells, the rate of repair lags behind the rate of mutation, which makes the cancer cells thrive and increase in numbers resulting in further destruction.
Plates include both oceanic crust and continental crust. Stable subduction zones involve the oceanic lithosphere of one plate sliding beneath the continental or oceanic lithosphere of another plate due to the higher density of the oceanic lithosphere.
Answer:
the fundamental unit of heredity
Explanation:
DNA is a double stranded helix structure. Each strand is made up of a string of nucleotides.
A gene is a region of DNA, usually tens of thousands of nucleotides long. At the simplest level, one gene encodes for one trait. Therefore, the gene can be described as the fundamental unit of heredity.
Genes work by coding for specific proteins, which carry out essentially all the functions in the cell.
Answer:
they both use photosynthesis
Explanation:
Answer:
they will lose their energy and die
download toppr for your studies using this link www.toppr.com/invitation/MUHA4RBX/?source=toppr
or use this refferal code after downloading toppr -- MUHA4RBX
