This is indeed true. But it depends on if u get a mutation after u have a kid or before
if u have a kid before mutation ur kid will most likely get a mutation as well.
if u have a kid after mutation rest assure ur kid will be safe... unless its contagious
Answer:
Glucose metabolism is impeded as the conversion of fructose-6-phosphate to fructose-2,6-bisphosphate in glycolysis is impossible after a knockout in mice.
On glucose levels, glucose levels rise as the feedback mechanism set in place to control the metabolism of glucose to ATP is absent.
Explanation:
GLUCOSE METABOLISM
PFK-2/FBPase-2 is an enzyme involved in regulating glycolysis and gluconeogenesis. It is found abundantly in organs such as the liver and heart.
It is a rate limiting enzyme critical in the catabolism of glucose which produces energy in the body.
Phosphofructokinase-2 (PFK2) catalyzes the conversion of fructose-6-phosphate to fructose-2,6-bisphosphate. PFK-2 is regulated by glucagon in the liver, epinephrine in muscle and by insulin, glucagon and epinephrine being hormones.
A knockout in mice, will conversely lead to an increased level of fructose-6-phosphate in glycolysis and with a lack of the rate limiting enzyme, glucose levels rise, leading to a consequential rise in glucose levels.
The rate limiting enzyme functions in the body's natural feedback mechanism to regulate the production of energy as well as the release of glucose for the production of energy.
Mutations<span> lead to </span>genetic variation<span> by making random changes in an organism's genetic code. This can </span>causes<span> a different protein to be created or none at all.</span>
25%,1:3 This is due to the heterozygous condition to produce on homozygous long and one homozygous short then two heterozygous short.ie 1:2:1,but short is dominant and heterozygous short ate taken to be short ,thus modifying the ratio to 1:3,1:3 is 25%:75%.therefore homozygous long is 25%.