Answer:
I don't know sorry need kalng points HAHAHAHAHAHA
When looking at a Neuromuscular Junction (NMJ), we know that neurotransmitters (NT) are released from the presynaptic cell and they then bind to the receptors that are located on the postsynaptic cell - this causes the effect of the NT being released.
So we are told that NT are still being released, however they are not having an effect. This would mean that they are probably being blocked by something - in this case, it seems that the neurotoxin is the culprit in the blocking of these receptors.
Therefore, if the NT cannot bind to the receptors on the postsynaptic cell, they are not going to have any effect, no matter how much NT is being released.
So the answer in this case is: The neurotoxin is most likely C) Blocking the receptors on the postsynaptic cell.
HI!
I am 99% sure cerebrum is your answer. Please tell me if I'm wrong.
Goo Day
Answer:
H+
Explanation:
Hemoglobin is the major protein of red blood cells. It has many exposed amino groups and carboxylic groups at its surface. These NH3 and COOH groups serve as weak acids and bases respectively and allow hemoglobin to serve as a buffer to maintain the pH of the RBC cytoplasm.
As the exposed amino groups of hemoglobin protein bind to the H+ ions, the free H+ concentration of the cytoplasm of RBC is reduced leading to a buffer action to maintain the pH.
Conjugation. I hope this helps!
