1950 I believe possibly 1970
Awareness context does the nurse determine this is closed awareness
<h3>What is pancreatic cancer ?</h3>
However, as the pancreatic cancer progresses and spreads, upper abdominal pain frequently begins to manifest, occasionally moving to the back. Following a meal or lying down, the pain might get worse. Jaundice, nauseousness, appetite loss, weight loss, exhaustion, sluggishness, and depression are some additional symptoms that could exist.
- Known risk factors for pancreatic cancer include smoking, diabetes, chronic pancreatitis, or inflammation of the pancreas, a family history of the disease, and a few genetic syndromes. Another contributing factor might be carrying extra weight that is unhealthy for your body.
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Changes in mental status referred as "delirium".
These also can be described as depression, dementia, and coma.
What is Change in Mental status?
- Change in Mental status results in life threatening situations.
- Generally, changes in consciousness can be divided into changes of arousal, the content of consciousness, or a combination.
- Hypoactivity can be described by tiredness and Arousal includes it.
- Depression results in personal withdrawal, slowed speech, or poor results of a cognitive test.
- Coma is a complete loosing of consciousness in which they don't respond.
To learn more about Mental status
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Answer:
b) blastic red blood cell (RBC).
Explanation:
In excess of 340 blood group antigens have now been described that vary between individuals. Thus, any unit of blood that is nonautologous represents a significant dose of alloantigen. Most blood group antigens are proteins, which differ by a single amino acid between donors and recipients. Approximately 1 out of every 70 individuals are transfused each year (in the United States alone), which leads to antibody responses to red blood cell <u>(RBC) alloantigens</u> in some transfusion recipients. When alloantibodies are formed, in many cases, RBCs expressing the antigen in question can no longer be safely transfused. However, despite chronic transfusion, only 3% to 10% of recipients (in general) mount an alloantibody response. In some disease states, rates of alloimmunization are much higher (eg, sickle cell disease). For patients who become alloimmunized to multiple antigens, ongoing transfusion therapy becomes increasingly difficult or, in some cases, impossible. While alloantibodies are the ultimate immune effector of humoral alloimmunization, the cellular underpinnings of the immune system that lead to ultimate alloantibody production are complex, including antigen consumption, antigen processing, antigen presentation, T-cell biology.
