DNA replication goes in the 5' to 3' direction because DNA polymerase acts on the 3'-OH of the existing strand for adding free nucleotides. DNA replication can take place both the directions chemically. ... But these nucleotide triphosphates hydrolyze spontaneously under aqeuous conditions.
Answer: There is presence of tumor.
Explanation: The adhesion of cells to extracellular matrix (EMC) through integrins ( cell-EMC binding molecules, which are collagens, laminins and fibronectin) causes the activation of kinases in the cytoplasm.
However, kinanes helps in controlling the epithelial cell differentiation and upholding the epithelial tissues. This is done by the addition of phosphate groups to a substrate protein which is termed Protein phosporylation. Then, the kinases direct the affairs of the cell and it's activities. For example, it determines the cell division, anabolic and catabolic activities of the cell, movement of ions between the cell and it's environment (signal transduction), protein functions and etc.
Conclusively, since the activities of the cell like cell division and protein functions is dictated by the kinase, reduction in cell division that gave rise to rapid growth is put on hold. Hence, the tumor is been suppressed.
Note: the binding of cell-EMC is regulated by Transforming Growth Factor (TGF) β.
Do you have the solutions you want me to compare and contrast? :)
There are
50 protons in tin
50 electrons in tin
And 69 neutrons
Hope this helps
The purpose of a bacterium's capsule is protection.
<h3>What use does the capsule of a bacteria serve?</h3>
White blood cells may eat and kill pathogens, but capsules could stop that from happening (phagocytosis). Although the precise mechanism for avoiding phagocytosis is unknown, it may occur because the presence of capsules makes the surface elements of bacteria more slippery, preventing the bacterium from being absorbed by phagocytic cells.
Capsules comprised of bacterial defenses and the host. The main function of the capsules of pathogenic bacteria is to prevent opsonophagocytosis or in the case of Gram-negative bacteria, complement-mediated lysis by shielding the bacterial surface from interactions with the host immune system.
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