Answer:
No, telomerase is not an oncogene. It prevents the senescence that would occur due to shortened telomeres, but the cell proliferation might still be mitogen-dependent.
Explanation
telomerase is not responsible for causing deregulation while oncogenes cause deregulation .
Telomeres length shorten after the cell division which stops them to divide again and cell die.
Telomerase prevents this decline in some kinds of cells, including stem cells, by lengthening telomeres, and the hope was that activating the enzyme could prevent senescence.
The effect differs. It could produce a different amino acid in the sequence because the corresponding codon has changed. It could also prevent the production of the originally intended sequence by changing one of the amino acids of a "start" codon (aka AUG) or extend the protein's sequence by modifying a "stop" codon (UAA, UAG, UGA or UGG), producing a new protein that might be useless or have different effects on the cell.
Answer:
Cruciferous
Explanation:
Their consumption is linked with lower rates of cancer and is believed to be associated with their sulfur containing phytochemicals
Extinct is correct. When no members of a species remain, the species is said to be extinct.
If it is the same dominance as usual it is homozygous
