Answer:
knirps (a gap gene)
wingless (a segment-polarity gene)
kruppel (a gap gene)
even-skipped ( a pair-rule gene)
hunchback (a gap gene)
hedgehog (a segment-polarity gene)
Explanation:
The gap, segment-polarity and pair-rule genes play central roles in controlling embryonic development of arthropods. In the first place, the gap genes are associated with the formation of contiguous body segments, thereby mutations in these genes result in gaps in the normal body plan of the embryo. For example, in <em>Drosophila melanogaster</em>, mutations in the <em>knirps, Krüppel </em>and<em> hunchback</em> genes result in deletion of body segments. These genes are also known to regulate segment polarity genes, which determine the polarity of the embryonic parasegments by modulating Wnt and Hedgehog signaling pathways. Finally, the pair-rule genes work together with gap genes to control embryonic development of alternating body segments.
Small, but normal variations in genes may produce proteins that work differently from those of other individuals ( friends or relatives). This may affect how we respond to different medicines. The extent to which these proteins functions varies from one individual to another. Example; certain painkillers only work when body proteins convert them from an inactive form to an active one. If these proteins work too fast, the drug will be eliminated from the body before it has time to work. To do their job the pain killers need to bind and modulate a target body protein, the receptor, such that if the target body protein is altered the pain killer might not be able to bind.
Answer:
A beacuse they can die without food
