Answer/Explanation: On Mercury temperatures can get as hot as 430 degrees Celsius during the day and as cold as -180 degrees Celsius at night.
Mercury is the planet in our solar system that sits closest to the sun. The distance between Mercury and the sun ranges from 46 million kilometers to 69.8 million kilometers. The earth sits at a comfy 150 million kilometers. This is one reason why it gets so hot on Mercury during the day.
The other reason is that Mercury has a very thin and unstable atmosphere. At a size about a third of the earth and with a mass (what we on earth see as ‘weight’) that is 0.05 times as much as the earth, Mercury just doesn’t have the gravity to keep gases trapped around it, creating an atmosphere. Due to the high temperature, solar winds, and the low gravity (about a third of earth’s gravity), gases keep escaping the planet, quite literally just blowing away.
Atmospheres can trap heat, that’s why it can still be nice and warm at night here on earth.
Mercury’s atmosphere is too thin, unstable and close to the sun to make any notable difference in the temperature.
Space is cold. Space is very cold. So cold in fact, that it can almost reach absolute zero, the point where molecules stop moving (and they always move). In space, the coldest temperature you can get is 2.7 Kelvin, about -270 degrees Celsius.
Sunlight reflected from other planets and moons, gases that move through space, the very thin atmosphere and the surface of Mercury itself are the main reasons that temperatures on Mercury don’t get lower than about -180 °C at night.
* More than 40 proteins and glycoproteins involved in the complement system are synthesized by the liver, macrophages, epithelial cells, they are present in the blood in plasmatic form, membrane, some have an enzymatic activity, regulator or membrane receptorThese are elements of the humoral innate immune response, they fight infections, purify immune complexes and apoptotic bodies.
<span>There are indeed three ways to activate the complement:</span>
Classical pathway: Activated by Immunoglobulins in immune complexes, aggregated Immunoglobulins, DNA, CRP, apoptotic bodies .......it involves nine fractions, starting with C1, then C4, C2, C3, to form a classical C5 convertase, then, activation of C5, C6, C7, C8, C9.
Alternative pathway: activated by polysaccharides (bacterial endotoxin), vascular wall poor in sialic acid, aggregated IgE ...C3b like is the first component in the alternate channel cascade, it will create an amplification loop, and form an alternative C5 convertase.
Lecithin pathway: Activated by mannose, fucose (carbohydrate of microorganisms)The first component is the complex MBL / MASP1 / MASP2: "mannose-binding protein": works according to the same principle as the complex C1 of the classical way (MASP2 cleaves the C4 and the rest of the cascade is equivalent to that of the classical way).
the three ways have the same outcome: A C5 convertase (formed by one of the pathways) cleaves C5 into C5a and C5b: C5b is deposited far from other fractions on the antigenic surface. The fixation of C5b in the cell is followed by that of C6, C7, C8, and C9 (9 molecules of C9): formation of the membrane attack complex (MAC) ==> Death of the cell by osmotic shock
The answer to this question is D have a nice day
