An infant born with esophageal atresia and tracheoesophageal fistula receives a prescription for internal feedings after corrective surgery. <u>An infant is born with esophageal atresia and tracheoesophageal fistula.</u>
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Esophageal atresia is a beginning defect in which a part of a baby's esophagus (the tube that connects the mouth to the stomach) no longer increases well. Esophageal atresia is a start defect of the swallowing tube (esophagus) that connects the mouth to the belly.
The precise cause of EA remains unknown, but it appears to have some genetic additives. as much as 1/2 of all infants born with EA have one or greater other beginning defects, together with: trisomy thirteen, 18, or 21. other digestive tract problems, such as intestinal atresia or imperforate anus.
Oesophageal atresia is a concept to be because of trouble with the development of the esophagus even as the child is in the womb, although it's not clear exactly why this takes place. The condition is extra, not unusual in babies of mothers who had too much amniotic fluid in being pregnant (polyhydramnios).
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Answer: Mutations in the GALT, GALK1, and GALE genes cause galactosemia.
Explanation: Galactosemia is a disorder that affects how the body processes a simple sugar called galactose. A small amount of galactose is present in many foods. It is primarily part of a larger sugar called lactose, which is found in all dairy products and many baby formulas. The signs and symptoms of galactosemia result from an inability to use galactose to produce energy.Classic galactosemia, also known as type I, is the most common and most severe form of the condition. If infants with classic galactosemia are not treated promptly with a low-galactose diet, life-threatening complications appear within a few days after birth. Affected infants typically develop feeding difficulties, a lack of energy (lethargy), a failure to gain weight and grow as expected (failure to thrive), yellowing of the skin and whites of the eyes (jaundice), liver damage, and abnormal bleeding. Other serious complications of this condition can include overwhelming bacterial infections (sepsis) and shock. Affected children are also at increased risk of delayed development, clouding of the lens of the eye (cataract), speech difficulties, and intellectual disability. Females with classic galactosemia may develop reproductive problems caused by an early loss of function of the ovaries (premature ovarian insufficiency). Galactosemia type II (also called galactokinase deficiency) and type III (also called galactose epimerase deficiency) cause different patterns of signs and symptoms. Galactosemia type II causes fewer medical problems than the classic type. Affected infants develop cataracts but otherwise experience few long-term complications. The signs and symptoms of galactosemia type III vary from mild to severe and can include cataracts, delayed growth and development, intellectual disability, liver disease, and kidney problems.
Answer:
Gametic cells.
4 cells.
Chromosomes number reduces to half.
Explanation:
Meiosis may be defined as a type of cell division in which a single parent cell divides to form the daughter cells. The sex cells or gametes (sperm and ova ) undergoes the meiosis division.
The single parent call divides into four daughter cells by the process of meiosis- I and meiosis- II. Meiosis II is similar to the process of mitosis.
Meiosis is also known as reduction division. The chromosome number reduces to half as compared with the parent cell. The haploid cells are produced in the meiosis.
Answer:
frankrubberrody . i tried to make it ryhem lol
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