Answer:
Brachialis
Explanation:
The Brachialis is an anterior skeletal arm muscle that extends from the medial arm to the superior area of the forearm. The brachialis fibers originate in the anterior part of the humerus and direct to the inferior part of the elbow, where they insert in the upper region of the ulna and distal radius. This muscle is an elbow flexor being used in every movement needed to approximate the anterior part of the forearm to the arm. It is a very thick and powerful muscle that acts with high speed.
The answer is
B) a flowerpot falling from a balcony.
This is the answer because potential energy is basically when something is standing still. Kinetic energy is the opposite, its when something is moving. So the flowerpot was still but something knocked it over and the potential energy was turned into kinetic energy. In option A, the kinetic energy is being turned into potential energy singe the car stopped moving. In option C, the cow is just storing potential energy and it is not being transformed into kinetic energy since the cow is not moving. Option D is not the answer because once again, this says the bike is slowing down, which means that the kinetic energy is decreasing and being turned into potential energy.
<span>he Streptococcus pneumoniae capsule is vital for virulence and may inhibit complement activity and phagocytosis. However, there are only limited data on the mechanisms by which the capsule affects complement and the consequences for S. pneumoniae interactions with phagocytes. Using unencapsulated serotype 2 and 4 S. pneumoniae mutants, we have confirmed that the capsule has several effects on complement activity. The capsule impaired bacterial opsonization with C3b/iC3b by both the alternative and classical complement pathways and also inhibited conversion of C3b bound to the bacterial surface to iC3b. There was increased binding of the classical pathway mediators immunoglobulin G (IgG) and C-reactive protein (CRP) to unencapsulated S. pneumoniae, indicating that the capsule could inhibit classical pathway complement activity by masking antibody recognition of subcapsular antigens, as well as by inhibiting CRP binding. Cleavage of serum IgG by the enzyme IdeS reduced C3b/iC3b deposition on all of the strains, but there were still marked increases in C3b/iC3b deposition on unencapsulated TIGR4 and D39 strains compared to encapsulated strains, suggesting that the capsule inhibits both IgG-mediated and IgG-independent complement activity against S. pneumoniae. Unencapsulated strains were more susceptible to neutrophil phagocytosis after incubation in normal serum, normal serum treated with IdeS, complement-deficient serum, and complement-deficient serum treated with IdeS or in buffer alone, suggesting that the capsule inhibits phagocytosis mediated by FcÎł receptors, complement receptors, and nonopsonic receptors. Overall, these data show that the S. pneumoniae capsule affects multiple aspects of complement- and neutrophil-mediated immunity, resulting in a profound inhibition of opsonophagocytosis.</span>
