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Naily [24]
2 years ago
13

Explain the agonist-to-antagonist spectrum of action of psychopharmacologic agents, including how partial and inverse agonist fu

nctionality may impact the efficacy of psychopharmacologic treatments.
Medicine
1 answer:
mash [69]2 years ago
4 0

Answer:

An agonist is a drug that binds to the receptor, producing a similar response to the intended chemical and receptor. Whereas an antagonist is a drug that binds to the receptor either on the primary site, or on another site, which all together stops the receptor from producing a response

Explanation:

hope it will help you

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In your own words explain the steps into creating a vaccine. <br><br> 55 points
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Put the fluid in the bottle.
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pubmed Long-term outcomes after stenting versus endarterectomy for treatment of symptomatic carotid stenosis: the International
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Stenting is an alternative to endarterectomy for treatment of carotid artery stenosis, but long-term efficacy is uncertain.

What is Stenting?

A stent is a small tube made of wire mesh. It holds open an artery and stays there indefinitely.

If a coronary artery (a blood vessel that supplies an accumulation narrows the heart muscle. Plaque is a fatty buildup that can lessen the blood flow. if there is less blood flow Chest pain may be caused by damage to the heart muscle.If a clot develops and entirely obstructs the a portion of the heart muscle's blood flowthe heart attack happens.Stents maintain coronary artery patency.and lower the risk of having a heart attack.

A stent is deployed by collapsing it and placing it over the

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Learn more about the Stenting with the help of the given link:

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3 0
1 year ago
Several studies have found that in the United States, their is a rising trend of obesity for people between the ages of 2 and 19
ale4655 [162]

I DID NOT COPY THIS. THIS IS ALL ORIGINAL: THIS TOOK 1/2 an hour to write. Hope this helps

Buried in recent headlines is the sobering fact that obesity is still on the rise in the United States. The latest federal data show that nearly 40 percent of American adults were obese in 2015–16, up from 34 percent in 2007–08. The prevalence of severe obesity also went up during the same period, from 5.7 percent to 7.7 percent. In 1985, no state had an obesity rate higher than 15 percent. In 2016, five states had rates over 35 percent.

Obesity is a grave public health threat, more serious even than the opioid epidemic. It is linked to chronic diseases including type 2 diabetes, hyperlipidemia, high blood pressure, cardiovascular disease, and cancer. Obesity accounts for 18 percent of deaths among Americans ages 40 to 85, according to a 2013 study challenging the prevailing wisdom among scientists, which had placed the rate at around 5 percent. This means obesity is comparable to cigarette smoking as a public health hazard; smoking kills one of five Americans and is the leading preventable cause of death in the United States.

The obesity crisis may be less dramatic than the opioid epidemic now gripping the nation, but it is just as deadly. Opioids accounted for around two-thirds of the 64,000 deaths related to drug overdose in 2016. Excess body weight leading to cancer causes about 7 percent of cancer-related deaths, or 40,000 deaths each year. This number doesn’t include deaths from the many other medical conditions associated with obesity. Obese people are between 1.5 to 2.5 times more likely to die of heart disease than people with normal body mass indices (BMIs).

There are also substantial economic losses associated with obesity. The medical costs of prevention, diagnosis, and treatment are estimated at $147 billion in 2008 dollars. Reduced economic productivity adds to these losses.  

Because rising obesity is attributed to an increase in caloric intake and a reduction in physical activity, many proposed solutions emphasize food and exercise. While such remedies may help in individual cases, policy solutions are almost certainly required to fight this alarming epidemic.    

Despite the thriving U.S. weight-loss market (worth $66 billion in 2017), there is no evidence that diet-related programs will curb obesity. Numerous studies indicate that diets are not effective in controlling or reversing weight gain. In fact, 50 percent of dieters weighed more than 11 pounds over their starting weight five years after their diet, according to one study.

A comprehensive discussion of the policy solutions to obesity is beyond the scope of this piece, and the jury is still out on which policies — targeting sugar consumption through taxes on sugary food and beverages, regulating nutrition labels to make them more effective in informing consumers, and limiting the advertising and marketing of unhealthy food, particularly to children — might curb the epidemic.

Taxing potentially harmful food products has shown some promise, though it is a politically fraught approach. A small number of American cities, including Philadelphia, Boulder, Colo., and Berkeley, Calif., have begun taxing sugar-sweetened beverages. Early results show that an excise tax on sugary drinks led to a 21 percent drop in their consumption in Berkeley.

Berkeley is hardly the epicenter of the obesity problem in the U.S., as the map shows, but the intervention’s success offers hope for the rest of the country. A peer-reviewed modeling study based on the Berkeley experience estimated that if a national sugar-sweetened beverages tax were implemented, it would result in lower national consumption of these drinks and reduced adult and child BMIs. Whether such a policy could be replicated nationally remains uncertain.2

When it comes to nutrition labels, there’s almost no evidence that these have an effect on consumers’ dietary intake, body weight, and overall health.  

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