Answer:
the differnt dna and rna strucures in the mammals cause differen tlooks they have also evolved from being in the wilf most of their lives
Explanation:
Lipogenesis takes place primarily in liver cells, but also in skeletal muscle and adipose tissue.It takes place<span> in the cytoplasm of your fat cells and liver cells. </span><span> Beta-oxidation is primarily done in the </span>Mitochondrial Matrix. Once the Acyl-carnitine comes into the mitchondrial matrix, <span>CPT-2 </span>enzyme takes the carnitine off and re-attaches to a CoA group,.
Answer: Eating excessive quantities of such molecules could deregulate this process, increasing methylation and repressing the expression of genes that should normally be expressed.
Explanation:
DNA methylation is one of the epigenetic mechanisms involved in the regulation of gene expression, because it is a process by which methyl groups are added to DNA.
Methylation then modifies the function of DNA when it is found in the promoter gene, it is essential for normal development and is associated with a number of key processes, including genomic imprinting, inactivation of the X chromosome, repression of repeating elements, aging, and carcinogenesis. Usually, <u>it acts to suppress gene transcription.</u>
For example, folic acid is essential for the rapid cell division that occurs during early fetal development and it also plays an important role in methylation and thus in gene regulation. <u>The metabolism of these vitamins is aimed at achieving adequate levels of DNA methylation, necessary for the cellular processes</u>. Eating excessive quantities of such molecules could deregulate this process, <u>increasing methylation and repressing the expression of genes that should normally be expressed</u>.
Temperature affects spermatogenesis, which functions best at body temperatures just a little lower than those.
<h3>Abstract:</h3>
To keep testicular temperatures below those of the body core, adequate thermoregulation is essential. The process of mammalian spermatogenesis and the resulting spermatozoa are negatively impacted by elevated testicular temperature. Therefore, sperm quality can be affected and the likelihood of infertility is increased by thermoregulatory dysfunction resulting in heat stress. This article reviews a variety of internal and external factors that may lead to testicular heat stress. We go into more detail on how heat stress affects the spermatogenesis process, the resulting epididymal spermatozoa, germ cells, and the alterations that result in the testis.
We also go over the chemical reactions of germ cells to heat exposure and potential processes, such as apoptosis, DNA damage, and autophagy, that could lead to heat-induced germ cell damage. Further explanation is provided for the intrinsic and extrinsic processes involved in the complex mechanism of germ cell death. These intricate apoptotic pathways ultimately result in the demise of germ cells.
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