Answer:
it is a catecholamine that stimulates alpha1 adrenergic receptors and, slightly, beta1 adrenergic receptors, without beta2 effect. It is the vasoconstrictor drug par excellence.
The way they talk and work together
An increase in alcohol consumption
Alcohol can boost GABA activity in the brain in one of two ways: either by acting on the presynaptic cell, which increases GABA release, or. It can influence the postsynaptic (i.e., signal-receiving) neuron, promoting the GABAA receptor's activation.
The brain circuits that regulate motivational processes, such as arousal, reward, and stress, are altered by chronic alcohol consumption. These modifications impact systems that use dopamine, opioid peptides, -aminobutyric acid, glutamate, and serotonin as signaling molecules as well as systems that control the brain's stress response. Frequent episodes of intoxication, obsession with alcohol, use of alcohol in spite of negative effects, and a need to seek out and use alcohol are all signs of alcoholism developing.
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Answer:
<u>Ulnar nerve</u> is also called funny bone and feels tingling sensation if <u>humerus </u>is bumbed. <u>Medial epicondyle</u> passes underneath this bony structure.
Explanation:
The funny bone isn't actually a bone, its actually a nerve that touches your humerus.It is the ulnar nerve, which travels down the arm posteriorly and eventually reaches the "cubital tunnel" and at this location (between the trochlea and medial epicondyle of humerus), ulnar nerve is called funny bone.
The nerve is relatively unprotected at this place.
A blow here may produce a tingling or a numbing sensation throughout the forearm and hand.
Hence, <u>Ulnar nerve</u> is also called funny bone and feels tingling sensation if <u>humerus </u>is bumbed. <u>Medial epicondyle</u> passes underneath this bony structure.
