Answer:
The correct answer will be:
1. Acetylcholine binds to receptors on the motor end plate
2. Chemically-regulated ion channels open, causing depolarization
3 End plate potentials trigger action potential(s).
4. Transverse tubules convey potentials into the interior of the cell
5. Ca++ is released from the sarcoplasmic reticulum
6. Ca++ binds to troponin, pulling on tropomyosin
7. Binding sites on actin are uncovered, allowing myosin to bind and carry out power strokes
8. Force increases.
9. Ca++ is pumped (re-sequestered) into the sarcoplasmic reticulum
10. Force decreases.
Explanation:
The muscle contraction is a highly controlled mechanism which begins at the neuromuscular junction with the release of the acetylcholine neurotransmitter. This neurotransmitter causes the depolarization of the membrane by binding to the receptors of the motor end plate which generates an action potential. This action potential is transmitted via T-tubules from sarcolemma to the sarcoplasmic reticulum.
The sarcoplasmic reticulum releases calcium ions which binds to the troponin protein. This troponin removes the protein tropomyosin from the actin causing the rotation of the tropomyosin exposing the binding sites for myosin. The myosin binds to the actin using energy from the ATP which pulls the actin causing contraction. Another ATP binds the myosin head which weakens the bond between myosin and actin which releases the myosin which decreases the force between them decreases and the muscles relax.
Answer:
Explanation:
avid experienced his first bout at around 12 years old. At first, it was uncontrollable crying, not wanting to get out of bed and go to school, and feelings of worthlessness. He was more sensitive than ever about being “left out.” But even with these feelings, he remained the consummate perfectionist. For example, anything less than an “A” in school would prove to David that he was a worthless person. Now at 18, David’s grades are dropping quickly. He can’t eat; he can’t sleep. The pain he suffers day
A patient has been diagnosed with angina. As he talks with the nurse, he asks several good questions about angina and seems able to concentrate on the explanations. He seems eager to learn how to manage his condition. What assessment can be made by the nurse <u>Hardiness</u>
<h3>What is
angina?</h3>
Chest pain or pressure, often known as angina or angina pectoris, is a sign of coronary heart disease and is typically brought on by insufficient blood flow to the heart muscle (myocardium).
A blockage or spasm of the arteries that feed blood to the heart muscle is typically the cause of angina. Anemia, abnormal cardiac rhythms, and heart failure are among additional factors. Atherosclerosis, a component of coronary artery disease, is the primary mechanism of coronary artery occlusion. The phrase means "a strangling feeling in the chest" and is derived from the Latin words angere ("to strangle") and pectus ("chest").
The degree of oxygen deprivation in the heart muscle and pain intensity are only weakly correlated.
To learn more about angina from the given link:
brainly.com/question/11568953
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Answer:
She is suffering from Gingivostomatitis.
Explanation:
The answer is skin
The first line of defence is your innate immune system. Level one of this system consists of physical barriers like your skin and the mucosal lining in your respiratory tract. The tears, sweat, saliva and mucous produced by the skin and mucosal lining are part of that physical barrier, too.
