Answer:
Explanation:
A convergent boundary (also known as a destructive boundary) is an area on Earth where two or more lithospheric plates collide. One plate eventually slides beneath the other, a process known as subduction. The subduction zone can be defined by a plane where many earthquakes occur, called the Wadati–Benioff zone.[1] These collisions happen on scales of millions to tens of millions of years and can lead to volcanism, earthquakes, orogenesis, destruction of lithosphere, and deformation. Convergent boundaries occur between oceanic-oceanic lithosphere, oceanic-continental lithosphere, and continental-continental lithosphere. The geologic features related to convergent boundaries vary depending on crust types.
Plate tectonics is driven by convection cells in the mantle. Convection cells are the result of heat generated by radioactive decay of elements in the mantle escaping to the surface and the return of cool materials from the surface to the mantle.[2] These convection cells bring hot mantle material to the surface along spreading centers creating new crust. As this new crust is pushed away from the spreading center by the formation of newer crust, it cools, thins, and becomes denser. Subduction begins when this dense crust converges with less dense crust. The force of gravity helps drive the subducting slab into the mantle.[3] As the relatively cool subducting slab sinks deeper into the mantle, it is heated, causing hydrous minerals to break down. This releases water into the hotter asthenosphere, which leads to partial melting of asthenosphere and volcanism. Both dehydration and partial melting occurs along the 1,000 °C (1,830 °F) isotherm, generally at depths of 65 to 130 km (40 to 81 mi).[4][5]
Some lithospheric plates consist of both continental and oceanic lithosphere. In some instances, initial convergence with another plate will destroy oceanic lithosphere, leading to convergence of two continental plates. Neither continental plate will subduct. It is likely that the plate may break along the boundary of continental and oceanic crust. Seismic tomography reveals pieces of lithosphere that have broken off during convergence
Answer: the cfu/g Gram-negative bacteria in the fecal sample is C = 3.0 × 10^3
Explanation:
We know that; Gram negative bacteria looks pale reddish in color under a light microscope from Gram staining.
therefore
There are 30 red bacterial colonies counted.
1 mL of from tube 1 was removed and added to tube with 99 mL saline (tube 2) dilution is 1/100.
transferred volume into the plate is 1 mL.
Now, we have to determine the cfu/g Gram-negative bacteria in the fecal sample
Formula to calculate CFU/g bacteria in fecal sample is expressed as;
C = n/(s×d )
where C is concentration (CFU/g)
, n is number of colonies
, s is volume transferred to plate
, d is dilution factor.
so we substitute
C = 30 / ((1/100) × 1)
C = 30 / 0.01
C = 3000
C = 3.0 × 10^3
THERFERE, the cfu/g Gram-negative bacteria in the fecal sample is C = 3.0 × 10^3
Cell membranes protect and organize cells. All cells have an outer plasma membrane that regulates not only what enters the cell, but also how much of any given substance comes in. Unlike prokaryotes, eukaryotic cells also possess internal membranes that encase their organelles and control the exchange of essential cell components. Both types of membranes have a specialized structure that facilitates their gatekeeping function.
Answer:
Explanation:
Molecular biology has enabled the identification of the mechanisms whereby inactive myostatin increases skeletal muscle growth in double-muscled (DM) animals. Myostatin is a secreted growth differentiation factor belonging to the transforming growth factor-β superfamily. Mutations make the myostatin gene inactive, resulting in muscle hypertrophy. The relationship between the different characteristics of DM cattle are defined with possible consequences for livestock husbandry. The extremely high carcass yield of DM animals coincides with a reduction in the size of most vital organs. As a consequence, DM animals may be more susceptible to respiratory disease, urolithiasis, lameness, nutritional stress, heat stress and dystocia, resulting in a lower robustness. Their feed intake capacity is reduced, necessitating a diet with a greater nutrient density. The modified myofiber type is responsible for a lower capillary density, and it induces a more glycolytic metabolism. There are associated changes for the living animal and post-mortem metabolism alterations, requiring appropriate slaughter conditions to maintain a high meat quality. Intramuscular fat content is low, and it is characterized by more unsaturated fatty acids, providing healthier meat for the consumer. It may not always be easy to find a balance between the different disciplines underlying the livestock husbandry of DM animals to realize a good performance and health and meat quality.