The malate-aspartate (M-A) shuttle, which moves reducing equivalents from the cytosol into mitochondria, offers an important mechanism to control glycolysis and lactate metabolism in the heart.
The membrane between the cytoplasm and the mitochondria is impermeable to the passage of electrons through it and as a result of glycolysis, the electron that is produced during glycolysis is transported for the purpose of gain of energy during oxidative phosphorylation.
In eukaryotes, the mitochondrion's semipermeable inner membrane serves as a conduit for the malate-aspartate shuttle, a biochemical pathway that transports electrons generated during glycolysis across the membrane for oxidative phosphorylation.
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