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galben [10]
1 year ago
7

74. when a patient is placed on warfarin therapy, the first factor that will be decreased is: a. factor ii b. factor v c. factor

vii d. factor viii quizlet
Medicine
1 answer:
ch4aika [34]1 year ago
3 0

When a patient is placed on warfarin therapy, the first factor that will be decreased C. factor VII is the first factor that will be decreased.

<h3>What does “warfarin therapy” mean?</h3>

The main aim of the warfarin medication is to lessen the blood’s propensity to clot, not to totally prevent it. As a result, while taking warfarin, the blood’s capacity to clot must be closely observed.

<h3>What is the most frequent side effect of warfarin treatment?</h3>

The most serious and frequent side effect of taking warfarin is bleeding. The potential benefit from thromboembolic disease prevention must be weighed against the risk of induced hemorrhagic side effects for any given patient.

<h3>How do I begin taking warfarin?</h3>

We advise the following during warfarin administration for the treatment of VTE: After receiving a diagnosis of VTE, start taking warfarin as soon as feasible, ideally the same day, in combination with UFH, LMWH, or fondaparinux.

To know more about warfarin therapy, visit:

brainly.com/question/13915413

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Tsu LV, Dienes JE, Dager WE. Vitamin K dosing to reverse warfarin based on INR, route of administration, and home warfarin dose
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Subsequent INR readings are influenced by the dose, method, and initial INR of vitamin K. For intravenous vitamin K doses of 2 mg or more, INR decrease is comparable. FFP preadministration has no effect on INR readings 48 hours or more after vitamin K administration.

What is Abstract of Vitamin K dosing to reverse warfarin based on INR, route of administration, and home warfarin dose in the acute/critical care setting?

  • Commonly, vitamin K is used to reverse the anticoagulant effects of warfarin. The ideal vitamin K dosage and delivery method that does not lengthen bridging therapy are still unclear.
  • To ascertain the elements affecting the level and pace of vitamin K-induced INR reversal in the acute/critical care setting.
  • 400 patients' charts from between February 2008 and November 2010 who got vitamin K to counteract the effects of warfarin were examined. International normalized ratios (INRs), intravenous or oral vitamin K doses, and whether or not fresh frozen plasma (FFP) was administered were among the information gathered. INRs were measured 12, 24, and 48 hours before vitamin K treatment.
  • At baseline, 12 hours, 24 hours, and 48 hours, respectively, intravenous vitamin K decreased INR more quickly than oral vitamin K (5.09, 1.91, 1.54, and 1.41 vs. 5.67, 2.90, 2.14, and 1.58). Subsequent INR values were impacted by baseline INR (p 0.001), method of administration (p 0.001), and vitamin K dosage (p 0.001). For intravenous vitamin K doses of 2 mg or more, there was a similar drop in INR. Home warfarin dose had no effect on INR responses to intravenous or oral vitamin K (p = 0.98 and 0.27, respectively). FFP had no effect on INR readings 48 hours later. Although larger vitamin K doses and longer anticoagulation bridge therapy appeared to be related, neither the incidence (p = 0.63) nor the duration (p = 0.61) were statistically significant.

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