Two points that are attached are the origin and insertion. The origin is the attachment joint that is usually at the proximal end of a bone, and the insertion is at the distal end of a joint.
This is contradictory because some teachers might teach it does and same do not. It protects people, so I would say true, but on what level does it truly protect them?
(C) Autoimmune destruction of B-cells of the pancreas is the etiological cause of this patient's symptoms.
The death of insulin-producing pancreatic beta-cells by autoreactive T cells characterizes type 1 diabetes (T1D), an autoimmune condition. Patients eventually need lifetime insulin treatment to maintain normal glycemic control when beta-cell loss causes insulin insufficiency and hyperglycemia.
Pancreatic B-cell autoimmunity is the cause of type 1 diabetes mellitus. Hyperglycemia, polyuria, increased thirst, weight loss, increased hunger, and nausea/vomiting are a few of the typical signs and symptoms of type 1 diabetes.
Elevated blood sugar levels and possible glucose present in the urine Along with other symptoms, diabetic ketoacidosis (as in this patient) might show up as ketone bodies in the urine and Kussmaul breathing.
Here is another question with an answer similar to this about type 1 diabetes: brainly.com/question/14823945
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Question correction:
A 12-year-old girl becomes comatose and is rushed to the hospital by her parents. She went to school feeling ill 2 days before the admission. She vomited that evening. Her vomiting persisted with only an 8-hour pause during sleep. She is breathing deeply and rapidly; her breath has a fruity odor. Her parents mention that her appetite has increased. She has also been drinking a lot of fluids; subsequently, she has been urinating more than normal. Urinalysis reveals 3+ glucose levels and 2+ ketone bodies. What is the etiological cause of this patient's symptoms?
A. Insulin resistance
B. Increase in counterregulatory hormones
C. Autoimmune destruction of B-cells of the pancreas
D. Post-Epstein-Barr virus infection
E. Autoimmune destruction of pancreatic acini cells