Answer: All their offsprings will be heterozygous for both traits.
Explanation: The offsprings of this cross will be heterozygous for the two traits. Each the offspring will have one dominant allele and one recessive allele for both traits. All the offsprings will have a genotype of BbEe. B is a dominant allele and b is a recessive allele while E is a dominant allele and b is a recessive allele.
Answer:
The lake was the result of an ice dam on the Clark Fork caused by the southern encroachment of a finger of the Cordilleran Ice Sheet into the Idaho Panhandle. It was the largest ice-dammed lake known to have occurred.
Hope this helps:)
Answer:
1 day.
Explanation:
From the given information:
The rate of mutation induced by RNA polymerase is:
Since the genome comprise of 19000 bp;
To complete a single replication, the viral cycle induce: 
However, in a single day; 10,000,000,000 viruses are reported to be replicated.
Hence, if a virus has 2 mutations; Then 10,000,000,000 virus will have:
SO, to round this up to whole number, it will take 1 day (s) to overcome the therapy.
Answer:
A person's genotype is represented as
b) DD, Dd, or dd
Lean persons may acquire a lipid profile with raised LDL- and HDL-cholesterol levels and reduced triglycerides when they follow carbohydrate-restricted diets (CRDs) (TGs). This lipid profile's intensity is correlated with BMI, therefore people with lower BMIs show greater rises in both LDL-C and HDL-C. Lean Mass Hyper-Responders (LMHR) is a subset of people who, while having normal pre-diet LDL-C, as shown by the inverse relationship between BMI and changes in LDL-C and HDL-C on CRD, respond to weight loss by increasing their HDL-C and LDL-C levels. revealed a substantial hyperlipidemic response to a CRD, with mean LDL-C and HDL-C levels rising to 320 and 99 mg/dL, respectively, in the context of mean TG of 47 mg/dL, as opposed to non-LMHR (mean values of 148 and 145 mg/dL, respectively). Again, with very normal pre-diet LDL-C and the absence of genetic evidence suggestive of familial hypercholesterolemia in those who have been examined, some LMHR may have LDL-C levels above 500 mg/dL.With carbohydrate restriction in lean individuals, the increased dependence on fat as a metabolic substrate drives increased hepatic secretion and peripheral uptake of TG contained within very low-density lipoproteins (VLDL), resulting in marked elevations of LDL-C and HDL-C and low TG, according to the Lipid Energy Model (LEM), which aims to explain this metabolic phenomenon. Here, we go through the LEM's main characteristics. We examine various lines of existing research that support the model and make recommendations for how to test its predictions.
To learn more about the lipid Energy Model here:-
brainly.com/question/17412429?referrer=searchResults
#SPJ4
