When looking at a Neuromuscular Junction (NMJ), we know that neurotransmitters (NT) are released from the presynaptic cell and they then bind to the receptors that are located on the postsynaptic cell - this causes the effect of the NT being released.
So we are told that NT are still being released, however they are not having an effect. This would mean that they are probably being blocked by something - in this case, it seems that the neurotoxin is the culprit in the blocking of these receptors.
Therefore, if the NT cannot bind to the receptors on the postsynaptic cell, they are not going to have any effect, no matter how much NT is being released.
So the answer in this case is: The neurotoxin is most likely C) Blocking the receptors on the postsynaptic cell.
Oof I don't think ppl like that (ŏ_ŏ)
The different degrees of sleep and wakefulness through which newborns cycle, ranging from deep sleep to great agitation, are called states of arousal. These states are regular sleep, irregular sleep, drowsiness, quiet alertness and crying. About eight to nine hours each of an infant's day is dedicated to regular and irregular sleep. So, most of the time of the infant is spent in these states. These are the states where the brain of an infant is developing the important connections for growth and survival. Crying is the means for infants to communicate their needs. Different type of cries would represent different needs of the infant.
Red giant or red supergaint (its the same thing)
